always sick

Discussion in 'Fibromyalgia Main Forum' started by Jittle, Jul 19, 2011.

  1. Jittle

    Jittle Member

    I am constantly getting some kind of infection: Sinus, ear, upper respiratory..... It seems if I am not on an antibotic I get sick all the time. I was just on one for a sinus infection. I finished it a few days ago, and woke up yesterday the infection moved into my chest. I get plenty of vitamins and eat realtivly well. I understand my immune system is probably not the best because of the FM but I don't know what to do. If I am not on an antibotic I get sick. I am going to rhuemy tomorrow and was going to ask if he had any ideas or knows a doc who can try to figure this out. Does anyone have any sugguestions or goes through this same thing?

  2. Nanie46

    Nanie46 Moderator


    Many people here have discovered that the actual root cause of their symptoms was a chronic infection, Borrelia burgdorferi (lyme disease), and some of the common coinfections that go with it such as Babesia, Bartonella, Ehrlichia, etc.

    It is common sense that there has to be a cause of your symptoms.

    Often the cause is infectious.

    Please keep and open mind and read this information. I welcome your questions.
  3. richvank

    richvank New Member

    Hi Jit.

    Your immune system is clearly not working as well as it should. You mention having FM, but could there be an ME/CFS component, too? The immune system is dysfunctional in most cases of ME/CFS, particularly the cell-mediated immune response, which is the one needed to combat viruses, fungi (including yeasts) and intracellular bacteria, including mycoplasma, chlamydia and rickettsia.

    In nearly all cases of ME/CFS that have been tested, there is a partial block in the methylation cycle, and usually glutathione depletion as well. These form a vicious circle, and that appears to be what makes ME/CFS chronic. This vicious circle also includes disruption of the folate metabolism.

    Glutathione and folate are very important for the cell-mediated immune response, and this vicious circle seems to me to be the cause of the immune dysfunction in this disorder.

    I suggest that people who suspect that they might have this vicious circle to run the methylation pathways panel offered by Health Diagnostics and Research Institute in New Jersey. Contact information and a guide for interpretation of the results are pasted below.

    If it turns out the there is such a vicious circle, there is a treatment that consists only of targeted nutritional supplements. In a clinical study carried out by Dr. Neil Nathan, this treatment was found to be of significant help to over two-thirds of the ME/CFS patients, most of whom also met the criteria for FM. The most recent version of this treatment is also pasted below.

    I am not financially affiliated with the lab test or the supplements. I hope this is helpful.

    Best regards,


    Methylation Pathways Panel

    This panel will indicate whether a person has a partial methylation cycle block and/or glutathione depletion. I recommend that this panel be run before deciding whether to consider treatment for lifting the methylation cycle block. I am not associated with the lab that offers this panel.

    The panel requires an order from a physician or a chiropractor. The best way to order the panel is by fax, on a clinician’s letterhead.

    Available from:

    Health Diagnostics and Research Institute
    540 Bordentown Avenue, Suite 2300
    South Amboy, NJ 08879
    Phone: (732) 721-1234
    Fax: (732) 525-3288

    Lab Director: Elizabeth Valentine, M.D.

    Dr. Tapan Audhya, Ph.D., is willing to help clinicians with interpretation of the panel by phone, or you can use the guide below:

    May 19, 2011

    Interpretation of Results of the Methylation Pathways Panel

    Richard A. Van Konynenburg, Ph.D.
    Independent Researcher
    (e-mail address removed by ProHealth Moderator)

    Disclaimer: The Methylation Pathways Panel is offered by the European Laboratory of Nutrients in the Netherlands and the Health Diagnostics and Research Institute in New Jersey, USA. I am not affiliated with these laboratories, but have been a user of this panel, and have written these suggestions at the request of Tapan Audhya, Ph.D., Director of Research for the Health Diagnostics lab, for the benefit of physicians who may not be familiar with this panel. My suggestions for the interpretation of results of the panel are based on my study of the biochemistry involved, on my own experience with interpreting panel results as part of the analysis of a fairly large number of cases of myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) over the past four years, and on discussion of some of the issues with Dr. Audhya. I am a researcher, not a licensed physician. Treatment decisions based on the results of applying this panel and its interpretation to individual cases are the responsibility of the treating physician.

    Application: In addition to being useful in analyzing cases of ME/CFS, this panel can also be usefully applied to cases of autism and other disorders that involve abnormalities in glutathione, methylation and the folate metabolism.

    The panel includes measurement of two forms of glutathione (reduced and oxidized), S-adenosylmethionine (SAMe), S-adenosylhomocysteine (SAH), adenosine, and seven folate derivatives.

    According to Dr. Audhya (personal communication), the reference ranges shown on the lab reports for each of these metabolites were derived from measurements on at least 120 healthy male and female volunteer medical students from ages 20 to 40, non-smoking, and with no known chronic diseases. The reference ranges extend to plus and minus two standard deviations from the mean of these measurements.

    Glutathione (reduced): This is a measurement of the concentration of the
    chemically reduced (active) form of glutathione (abbreviated GSH) in the blood
    plasma. The reference range is 3.8 to 5.5 micromoles per liter.

    Glutathione plays many important roles in the biochemistry of the body, including serving as the basis of the antioxidant enzyme system, participating in the detoxication system, and supporting the cell-mediated immune response, all of which exhibit deficits in CFS. The level of GSH in the plasma is likely to be more reflective of tissue intracellular glutathione status than the more commonly and more easily measured red blood cell or (essentially equivalent) whole blood glutathione level, which is about three orders of magnitude greater, because red blood cells are normally net producers of glutathione. Also, knowledge of the level of the reduced form, as distinguished from total (reduced plus oxidized) glutathione, which is more commonly measured, is more diagnostic of the status of glutathione function.

    In order to be able to approximate the in vivo level of reduced glutathione when blood samples must be shipped to a lab, it is necessary to include special enzyme inhibitors in the sample vials, and these are included in the test kit supplied by these two laboratories.

    Most people with chronic fatigue syndrome (PWCs), but not all, are found to have values of GSH that are below the reference range*. This means that they are suffering from glutathione depletion. As they undergo treatment to lift the partial methylation cycle block, this value usually rises into the normal range over a period of a few months. I believe that this is very important, because
    glutathione normally participates in the intracellular metabolism of vitamin B12, and if it is low, a functional deficiency of vitamin B12 results, and insufficient methylcobalamin is produced to support methionine synthase in the methylation cycle. In my view, this is the mechanism that causes the onset of ME/CFS. This functional deficiency is not detected in a conventional serum B12 test, but will produce elevated methylmalonate in a urine organic acids test. In my opinion, many of the abnormalities and symptoms in ME/CFS can be traced directly to glutathione depletion.

    Anecdotal evidence suggests that PWCs who do not have glutathione depletion do have abnormalities in the function of one or more of the enzymes that make use of glutathione, i.e. the glutathione peroxidases and/or glutathione transferases. This may be due to genetic polymorphisms or DNA adducts on the genes that code for these enzymes, or in the case of some of the glutathione peroxidases, to a low selenium status.

    Glutathione (oxidized): This is a measurement of the concentration
    of the oxidized form of glutathione (abbreviated GSSG) in the blood
    plasma. The reference range is 0.16 to 0.50 micromoles per liter.

    Normally, oxidized glutathione in the cells is recycled back to reduced glutathione by glutathione reductase, an enzyme that requires vitamin B2 and NADPH. If this reaction is overwhelmed by oxidative stress, the cells export excess GSSG to the plasma. In some (but not all) PWCs, GSSG is elevated above the normal
    range, and this represents oxidative stress. It is more common in CFS to see this level in the high-normal range. This value may increase slightly under initial treatment of a partial methylation cycle block.*

    Ratio of Glutatione (reduced) to Glutathione (oxidized): This is not shown explicitly on the panel results, but can be calculated from them. It is a measure of the redox potential in the plasma, and reflects the state of the antioxidant system in the cells. The normal mean value is 14. PWCs often have a value slightly more than half this amount, indicating a state of glutathione depletion and oxidative stress. This ratio has been found to increase during treatment of a partial methylation cycle block.*

    S-adenosymethionine (RBC): This is a measure of the concentration of S-adenosylmethionine (SAMe) in the red blood cells. The reference range is 221 to 256 micromoles per deciliter.

    SAMe is produced in the methylation cycle and is the main supplier of methyl (CH3) groups for a large number of methylation reactions in the body, including the methylation of DNA and the biosynthesis of creatine, carnitine, coenzyme Q10, melatonin and epinephrine. This measurement is made in the red blood cells because the level there reflects an average over a longer time and is less vulnerable to fluctuations than is the plasma level of SAMe.

    Most PWCs have values below the reference range, and treatment raises the value.* A low value for SAMe represents a low methylation capacity, and
    in CFS, it usually appears to result from an inhibition or partial block of the enzyme methionine synthase in the methylation cycle. Many of the abnormalities in CFS can be tied to lack of sufficient methylation capacity.

    S-adenosylhomocysteine (RBC): This is a measure of the
    concentration of S-adenosylhomocysteine (SAH) in the red blood cells. The reference range is 38.0 to 49.0 micromoles per deciliter.

    SAH is the product of the many methyltransferase reactions that utilize SAMe as a source of methyl groups. In CFS, its value ranges from below the reference range to above the reference range. Values appear to be converging toward the reference range with treatment.

    Sum of SAM and SAH: When the sum of SAM and SAH is below about 268
    micromoles per deciliter, it appears to suggest the presence of
    upregulating polymorphisms in the cystathionine beta synthase (CBS)
    enzyme, though this may not be true in every case. For those considering following the Yasko treatment program, this may be useful information.

    Ratio of SAM to SAH: A ratio less than about 4.5 represents low
    methylation capacity. Both the concentration of SAM and the ratio of
    concentrations of SAM to SAH are important in determining the
    methylation capacity, because they affect the rates of the methyltransferase reactions.

    Adenosine: This is a measure of the concentration of adenosine in the
    blood plasma. The reference range is 16.8 to 21.4 x 10(-8) molar.

    Adenosine is a product of the reaction that converts SAH to homocysteine. It is also exported to the plasma when mitochondria develop a low energy charge, so that ATP drops down to ADP, AMP, and eventually, adenosine. Adenosine in the plasma is normally broken down to inosine by the enzyme adenosine deaminase.

    In some PWCs adenosine is found to be high, in some it is low, and in some it is in the reference range. I don't yet understand what controls the adenosine level in these patients, and I suspect that there is more than one factor involved. In most PWCs who started with abnormal values, the adenosine level appears to be moving into the reference range with methylation cycle treatment, but more data are needed.

    5-CH3-THF: This is a measure of the concentration of 5L-methyl
    tetrahydrofolate in the blood plasma. The reference range is 8.4 to 72.6 nanomoles per liter.

    This form of folate is present in natural foods, and is normally the most abundant form of folate in the blood plasma. It is the form that serves as a reactant for the enzyme methionine synthase, and is thus the important form for the methylation cycle. It is also the only form of folate that normally can enter the brain. Its only known reactions are the methionine synthase reaction and reaction with the oxidant peroxynitrite.

    When there is a partial block in methionine synthase, 5L-CH3-THF drains from the cells into the blood plasma by the so-called “methyl trap” mechanism. As other forms of folate are converted to 5L-CH3-MTF, this mechanism depletes the cells of folates in general.

    Many PWCs have a low value of 5L-CH3-MTF, consistent with a partial block in the methylation cycle. Most methylation treatment protocols include supplementation with 5L-CH3-MTF, which is sold over-the-counter as Metafolin, FolaPro, or MethylMate B (trademarks), and in the prescription “medical foods” supplied by PamLab, including Deplin, CerefolinNAC and Metanx. There are some others on the market that include both racemic forms (5L and 5R) of this folate.

    When methylation treatment is used, the level of 5-CH3-THF rises in nearly every PWC. If the concentration of 5-CH3-THF is within the reference range, but either SAM or the ratio of SAM to SAH is below the reference values, it suggests that there is a partial methylation cycle block and that it is caused by inavailability of sufficient bioactive B12, rather than inavailability of sufficient folate. A urine organic acids panel will show elevated methylmalonate if there is a functional deficiency of B12. I have seen this combination frequently, and I think it demonstrates that the functional deficiency of B12 is the immediate root cause of most cases of partial methylation cycle block. Usually glutathione is low in these cases, which is consistent with such a functional deficiency. As the activity of the methylation cycle becomes more normal, the demand for 5-CH3-THF will likely increase, so including it in the treatment protocol, even if not initially low, will likely be beneficial.

    10-Formyl-THF: This is a measure of the concentration of 10-formyl
    tetrahydrofolate in the blood plasma. The reference range is 1.5 to 8.2 nanomoles per liter.

    This form of folate is involved in reactions to form purines, which form part of RNA and DNA as well as ATP. It is usually on the low side in PWCs, likely as a result of the methyl trap mechanism mentioned above. This deficiency is likely the reason for some elevation of mean corpuscular volume (MCV) and mean corpuscular hemoglobin (MCH) often seen in PWCs. This deficit may also impact replacement of cells lining the gut, as well as white blood cells.

    5-Formyl-THF: This is a measure of the concentration of 5-formyl
    tetrahydrofolate (also called folinic acid) in the blood plasma. The reference range is 1.2 to 11.7 nanomoles per liter.

    This form is not used directly as a substrate in one-carbon transfer reactions, but it can be converted into other forms of folate, and may serve as a buffer form of folate. Most but not all PWCs have a value on the low side. It is one of the
    supplements in some methylation protocols. It can be converted to 5L-CH3-THF in the body by a series of three reactions, one of which requires NADPH, and it may also help to supply other forms of folate until the methionine synthase reaction comes up to more normal activity.

    THF: This is a measure of the concentration of tetrahydrofolate in
    the blood plasma. The reference range is 0.6 to 6.8 nanomoles per liter.

    This is the fundamental chemically reduced form of folate from which several other reduced folate forms are synthesized, and thus serves as the “hub” of the folate metabolism. THF is also a product of the methionine synthase reaction, and participates in the reaction that converts formiminoglutamate (figlu) into glutamate in the metabolism of histidine. If figlu is found to be elevated in a urine organic acids panel, it usually indicates that THF is low. In PWCs it is lower than the mean normal value of 3.7 nanomoles per liter in most but not all PWCs.

    Folic acid: This is a measure of the concentration of folic acid in
    the blood plasma. The reference range is 8.9 to 24.6 nanomoles per liter.

    Folic acid is a synthetic form of folate, not found in nature. It is added to food grains in the U.S. and some other countries in order to lower the incidence of neural tube birth defects, including spina bifida. It is the oxidized form of folate, and therefore has a long shelf life and is the most common commercial folate supplement. It is normally converted into THF by two sequential reactions catalyzed by dihydrofolate reductase (DHFR), using NADPH as the reductant. However, some people are not able to carry out this reaction well for genetic reasons, and PWCs may be depleted in NADPH, so folic acid is not the best supplemental form of folate for these people.

    Low values suggest folic acid deficiency in the current diet. High values, especially in the presence of low values for THF, may be associated with inability to convert folic acid into reduced folate readily, such as because of a genetic polymorphism in the DHFR enzyme. They may also be due to high supplementation of folic acid.

    Folinic acid (WB): This is a measure of the concentration of folinic acid in the whole blood. The reference range is 9.0 to 35.5 nanomoles per liter.

    See comments on 5-formyl-THF above. Whole blood folinic acid usually tracks with the plasma 5-formyl-THF concentration.

    Folic acid (RBC): This is a measure of the concentration of folic acid in the red blood cells. The reference range is 400 to 1500 nanomoles per liter.

    The red blood cells import folic acid when they are initially being formed, but during most of their lifetime, they do not normally import, export, or use it. They simply serve as reservoirs for it, giving it up when they are broken down.

    Many PWCs have low values of this parameter. This can be caused by a low folic acid status in the diet over the previous few months, since the population of RBCs at any time has ages ranging from zero to about four months. However, in CFS it can also be caused by oxidative damage to the cell membranes, which allows folic acid to leak out of the cells. Dr. Audhya reports that treatment with omega-3 fatty acids has been found to raise this value over time in one cohort.

    If anyone finds errors in the above suggestions, I would appreciate being notified at

    * Nathan, N., and Van Konynenburg, R.A., Treatment Study of Methylation Cycle Support in Patients with Chronic Fatigue Syndrome and Fibromyalgia, poster paper, 9th International IACFS/ME Conference, Reno, Nevada, March 12-15, 2009. (

    March 30. 2011

    IN CHRONIC FATIGUE SYNDROME—March 30, 2011 Revision
    Rich Van Konynenburg. Ph.D.
    (Based on the full treatment program
    developed by Amy Yasko, Ph.D., N.D.
    which is used primarily in treating autism [1])


    1. General Vitamin Neurological Health Formula [2]: Start with ¼ tablet and increase dosage as tolerated to 2 tablets daily
    2. Hydroxy B12 Mega Drops [3]: 2 drops under the tongue daily
    3. MethylMate B [4]: 3 drops under the tongue daily
    4. Folinic acid [5]: ¼ capsule daily
    5. Phosphatidyl Serine Complex [6]: 1 softgel capsule daily (or lecithin, see below)

    All these supplements can be obtained from
    The fourth supplement comes in capsules that contain 800 mcg. It will be necessary to open the capsules, dump the powder onto a flat surface, and separate it into quarters using a knife to obtain the daily dose. The powder can be taken orally with water, with or without food.
    These supplements can make some patients sleepy, so in those cases they take them at bedtime. In general, they can be taken at any time of day, with or without food.
    Phosphatidyl serine can lower cortisol levels. Patients who already have low evening cortisol levels may wish to substitute lecithin [7] (at one softgel daily) for supplement number 5 above. Lecithin is also available from
    For those allergic to soy, lecithin from other sources is available.
    GO SLOWLY. As the methylation cycle block is lifted, toxins are mobilized and processed by the body, and this can lead to an exacerbation of symptoms. IF THIS HAPPENS, try smaller doses, every other day. SLOWLY work up to the full dosages.
    Although this treatment approach consists only of nonprescription nutritional supplements, a few patients have reported adverse effects while on it. Therefore, it is necessary that patients be supervised by physicians while receiving this treatment.

    [1] Yasko, Amy, Autism, Pathways to Recovery, Neurological Research Institute, 2009, available from or Amazon.
    [2] General Vitamin Neurological Health Formula is formulated and supplied by Holistic Health Consultants LLC.
    [3] Hydroxy B12 Mega Drops is a liquid form of hydroxocobalamin (B12), supplied by Holistic Health Consultants. 2 drops is a dosage of 2,000 mcg.
    [4] MethylMate B is a liquid form of (6s)-methyltetrahydrofolate supplied by Holistic Health Consultants, based on Extrafolate S, a trademark of Gnosis S.P.A. 3 drops is a dosage of 210 mcg.
    [5] Folinic acid is 5-formyltetrahydrofolate. ¼ capsule is a dosage of 200 mcg.
    [5] Phosphatidyl Serine Complex is a product of Vitamin Discount Center. 1 softgel is a dosage of 500 mg.
    [7] Lecithin is a combination of phospholipids without phosphatidylserine. One softgel is a dosage of 1,200 mg.

  4. richvank

    richvank New Member

    Hi, Jam.

    Yes, grapeseed extract is an excellent supportive antioxidant for glutathione. It will take some of the oxidative stress load off glutathione while not using it for recycle, as some other antioxidants do.

    I know it's worked well for you, and you have delivered a very consistent message about this for a long time.

    Best regards,

  5. mbofov

    mbofov Active Member

    I had chronic sinus and upper respiratory infections for many years, was almost always sick, though did not take antibiotics all the time because they are so rough on the body and digestive tract, etc.

    A couple of things that helped me were:

    1. I discovered I had a mild allergy to my cat! I should have looked at this sooner because my mother and one of my sisters were allergic but I never had the sudden severe symptoms they had. I did some allergy clearing and that helped a lot. (for the allergy clearing technique, google "allergone allergy correction method" - it involves a little muscle testing and tapping on meridian points - it's simple to do and sounds a little strange, but it really works. I did it on my sister who has a severe cat allergy (as well as several other allergies) and she was able to take my parents' cat when they couldn't keep it any more - before if she just was around it or handled it a little she would have severe symptoms and now hardly reacts at all to the cat. We had to redo the clearing about 6 months after the initial one)

    2. Also, discovered I had a severe dust mite allergy, through testing at an allergist's office. So I had to take remedial action - get a dust mite proof mattress cover, special pillows and covers. Because of CFS, I can't vacuum or dust nearly as much as I should, but the covers do help. We also did allergy clearing for dust mites.

    3. I started taking something called Lactoferrin with Colostrum Plus by Symbiotics a couple of months ago and believe this is boosting my immune system too. It also helps the gut which is crucial for immune system health.

    4. In additoin to regular vitamins, also take 5,000 IU of vitamin D3 daily, and extra vitamin C when I get sick.

    I still get sick every time I crash, they go hand in hand, but I get over being sick quicker now, and don't get sick as often in between crashes.

    So my immune system is still compromised due to CFS, but it's definitely stronger than before.

    I strongly suggest you get tested for allergies of all kinds - any animals you're around, dust mites, and foods. You might try cutting out wheat and dairy, which are extremely common allergens, and see if you notice any difference.

    I think Rich has a good suggestion re the methylation protocol. His protocol has helped many people although it didn't help me. I'm doing a similar protocol devised by Freddd on the Phoenix Rising board, and that is helping my energy, although I still crash. I wish someone could figure out what causes this crashing!

    Anyways, good luck -

  6. luigi21

    luigi21 Member

    i'll keep it simple jittle, i just had a water infection, followed by throat infection which went to my chest and turned into a chest infection, for me when i get one thing especially anything envolving ear nose throat, i usally end up with chest infections, dont know whether its because i dont produce excuse the crudeness 'snot' like others and so it drips down the back of my throat and causes a chest infection (common in sinus problems). the doctor gives me 250mg antibiotics, and yet sometimes i have to go back for 500mg. i do get complications as i have asthma. Good suggestion as ive already read here are probiotics, because antibiotics kill of the good bacteria as well as the bad, making you vunerable, so get some of them. putting vaseline, or vicks vapour rub, inside you nasal canal may make you less vunerable to infection as thats were bacteria infection usually takes hold, and of course washing your hands especially around sick people, or kids (they get sick alot).

    all the best

  7. Mikie

    Mikie Moderator

    If one suffers chronic sinus infections, it may be time to have an ENT do a scan of the sinuses. Polyps will cause obstructions in sinus drainage and the mucus will become infected. This can indeed lead to respiratory infections in the lungs. I even had the klebsiella bacteria from my sinus and chest infection go to my bladder and it was infected for a year. PWC have a difficult time overcoming infections. Sometimes, our weak immune systems just about give up trying to fight them and simply ignore them. That is when they become systemic and chronic. Taking ABX helps but doesn't address the problem.

    I have used the methylation protocol and the undenatured whey sold here to try to help my immune system and my whole body. I am also having to take the ABX and Acyclovir because I haven't been able to shake these chronic infections which are trying to reactive. I recently seemed to have picked up Sjogren's Syndrome and it has really taken its toll. I am fighting this on as many fronts as I can. I think I'll add transfer factors as they helped me beat my infections back into latency some years back.

    BTW, if you have any kind of infection, wash your hands both before and after going to the bathroom. You can contaminate the toilet paper with germs on your hands and if it can find a way into the urinary tract, it will.

    Love, Mikie
  8. Jittle

    Jittle Member

    So I told the doc yesterday. I asked hiim if he could help or could refer me to someone who could. right know he is starting off with blood work (running a lot of new and old tests again) I know for sure hs is going to run D, Lyme again, Lupus again, and something specific just for my immune system (if I could only spell it, That would be the day). and like 5 or 6 more but I forget for what. If that immune tets comes back I would then go see a specialist.

    GSE: I did start taking it, but read that it could interact (cause not to work as well) with my muscle relaxer. I may try it for a few days to see if I notice anything, and if I feel ok will continue.

    I am a crazy hand washer and sanitizer. Hubby says I am becoming OCD. Maybe he's right cause he is barley sick. And I change my toothbrush every time.

    Oh, Doc asked if I have always been llike this. I told him no, just for the past year. I said before that I never got sick, and if I did it was for like a day then I would feel better. Oh the days before FM

    I did read all of your posts. Thank you for the suggestion. They are great.

  9. mbofov

    mbofov Active Member

    I'm glad to hear I'm not the only crazy one! ;D To be honest, I don't care how crazy something sounds - if it works, I'm in.

    I did try cell food several years ago and had a very strong detox reaction, could not tolerate it. I don't think I went as low as 1/4 drop though. I still have it and may try it again some time.

    Right now I'm doing Freddd's B12/methylation protocol (on the Phoenix Rising board), and it is helping some with energy, but still crash.

    So my next plan involves systemic enzymes which I've read may help with the crashing. Unfortunatley they can also cause detoxing (it seems almost everything does!) but do plan to start them in about a week. I'm a little stronger than before, thanks to Freddd's protocol, so hope I can tolerate them better.

    Thanks for your suggestion - I'll add it to my list --

  10. Janalynn

    Janalynn New Member

    Funny because I, like a lot of people w/Fibro actually don't get sick very often. (Knock on wood) Maybe there is something else going on like everyone else suggested, Rich included.
    I think CFIDS has more to do with your immune system than Fibro itself does. OR, your illnesses could be totally unrelated to Fibro.

    Whatever it is, I hope you can figure it out because on top of everything else, being sick is no fun!