OK, since I cant post the link to the article, I will post the important parts of it. This was the scariest: Chapter One Some CFS Patients May Be "Non-HIV AIDS" Cases A disturbing announcement was made at the July 1992 international AIDS conference held in Amsterdam: Several people with symptoms of AIDS, but who had no evidence of infection with either HIV-1 or HIV- 2 (the viruses generally believed, at the time, to cause AIDS), had been identified by the U.S. Centers for Disease Control. A few weeks later, in early September, Newsweek made an even more shocking announcement: that Chronic Fatigue Syndrome researcher Dr. Paul Cheney had in his practice 20 CFS patients who had the same immune system deficiencies as the non-HIV AIDS cases revealed at the Amsterdam conference. What wasn't known to most observers was that one of the researchers who had first said publicly that he was aware of such cases, Dr. Sidhur Gupta of the University of California, Irvine, is himself a Chronic Fatigue Syndrome researcher. The fact that some CFS patients were developing exactly the same immune system problems as AIDS patients, however, raised the questions not only of what was causing that immune system destruction but also of the relationship that exists between the two syndromes. The hallmark of the HIV-negative AIDS cases, as established by the Centers for Disease Control, is a depletion of a type of immune system cell called T4 (or CD4) cells. The T4 cells of AIDS patients can fall to very low levels and, although recent studies have suggested that there is no real correlation with health status, a decreasing T4 cell count is generally viewed as a sign of worsening disease. The CDC decided to call the HIV-negative, AIDS-like disease "ICL" (an abbreviation for "idiopathic CD4-positive T-lymphocytopenia," which simply means an unexplained depletion of T4 cells). Most healthy people have a T4 cell count of approximately 1,000; a T4 cell count below 800 is considered abnormal. In order to be diagnosed with ICL, a person must have a T4 cell count of less than 300. One of the most puzzling things about the ICL cases to AIDS researchers -- other than the fact that they didn't have HIV -- is that most of the patients do not fit into recognized AIDS "risk behavior" categories; that is, they were not gay men, IV drug users, or the sexual partners of people in those risk groups. These cases may, in fact, be dramatic evidence that federal officials have not told the public the whole truth about the nature and the full scope of the AIDS epidemic. The mystery of what role HIV actually plays in causing the immune system deterioration and symptoms seen in AIDS deepened in early October 1992. The British medical journal The Lancet reported that five people had received blood from a man later found to be infected with HIV; however, ten years later, the five transfusion recipients as well as the original, HIV-positive blood donor remained free of AIDS symptoms and were apparently healthy. The Australian researchers who reported those cases concluded that these six people were infected with a non-disease-causing strain, or type, of HIV. The link between the immune system dysfunction seen in AIDS and in CFS was made explicit in early 1993 when government scientists admitted that CFS patients, like AIDS patients, suffer a decline in T4 cells. The government's leading CFS researcher, Dr. Stephen Straus at the National Institute of Allergy and Infectious Diseases, published a research paper in The Journal of Clinical Immunology in which a decrease in the number of T4 cells in CFS patients was documented. Dr. Straus proposed a novel mechanism to explain the loss of T4 cells in CFS patients: The T4 cells of CFS patients were not depleted, as they were in AIDS patients, according to Dr. Straus; they were just hiding in organ tissues. Unfortunately, Dr. Straus was unable to produce any evidence to support this theory (and still has not done so). Dr. Straus did not suggest that any of his CFS patients had T4 cell counts so low that they could be identified as ICL patients. Meanwhile, Dr. Cheney, in addition to announcing that some of his CFS patients had low enough T4 cell counts to be considered non- HIV AIDS cases, reported that as many as 40 percent of his CFS patients also had a close associate with an illness similar to CFS. This information -- along with the mystery of why AIDS could develop without HIV infection and why HIV infection does not always lead to AIDS -- raised the possibility that a virus or bacteria that spreads more easily than HIV could be attacking people's immune systems. OK, just the fact that it says its from 10 years ago discredits it somewhat. Anyone else?