Homeopathic Treatment of LymeVery Interesting Article

Discussion in 'Lyme Disease Archives' started by wrthster, Jul 15, 2007.

  1. wrthster

    wrthster New Member

    Most of us with Lyme never consider Homeopahty to treat it. For those who can not tollerate antibiotics or choose alternative methods should really look at this. It is very different from herbs. I welcome your feedback.

    Homeopathy and Lyme Disease
    by Ronald D. Whitmont, M.D.

    Abstract: The Demographics and Microbiology of Lyme disease and several other zoonotic infections are reviewed. Case studies utilizing the classical homeopathic system of assessment and treatment are presented. The discussion focuses on the strengths and limitations of the classical homeopathic and conventional allopathic models in the management of Lyme disease.
    Key words: Classical Homeopathy, Lyme Disease, Zoonotic Infections, Complementary and Alternative Medicine


    BACKGROUND
    Making the diagnosis of Lyme disease today has become increasingly complicated. Many physicians feel that Lyme is being constantly overdiagnosed and that too m any cases with so called soft evidence are being treated inappropriately.(1,2) (By soft evidence, I refer to a range of vague complaints running from chronic fatigue through fibromyalgia and depression.) On the other hand, there is a growing population that suffers from very real physical and emotional symptoms, but have found it difficult to prove that they have any identifiable disease despite the fact that, in many cases, all their symptoms date back to a tick bite.

    Medical research and popular literature reflect an ever-increasing number of reports on this topic.(3,4) In addition to the dilemma of making the correct diagnosis, the question of appropriate, effective management and cure has also become increasingly controversial. There are an increasing number of cases labeled as recurrent an d chronic Lyme disease, as well as cases of post Lyme disease syndrome. This all occurs despite the prolonged use of conventional antibiotic therapy according t o the standard of care. Almost every known infectious agent has demonstrated at least some degree of antibiotic resistance and this is probably true with Lyme disease.(5,6,7,8,9) Complementary and alternative medicine (CAM) and, particularly, Classical Homeopathy, is poised to offer viable alternatives and solutions to this modern dilemma in the case of Lyme disease and many other infectious problems.

    Demographics:
    Lyme Disease is not a common illness, but it is the most common vector borne disease in the United States today with an overall incidence of 6.2 per 100,000 population. The incidence, even in endemic areas, is estimated to be less than 1%. And 90% of all U.S. Cases occur in Connecticut, Massachusetts, New York, Rhode Island, New Jersey, Pennsylvania, Delaware, Wisconsin and Maryland. Eighty per cent of these cases are in the Northeast United States.(1) Only five states reported no Lyme disease in 1996: Alaska, Arizona, Colorado, Montana and South Dakota Lyme disease was first described in the mid 1970s near Old Lyme, Connecticut. It is not known whether it represents a new disease agent that has suddenly evolved, or whether it has been in existence for many years but only recently emerged from the fabric of zoonotic illnesses as a result of environmental stressors and shifts in the balances of host populations. Whatever its origins, it appears to be enjoying tremendous publicity as an ever increasing number of individuals become exposed to and demonstrate active infection. In 1996, over 16,000 cases of Lyme disease were reported to the Center for Disease Control. This represents a 41% increase since 1995.

    There are many illnesses and syndromes that are commonly misdiagnosed as Lyme Disease, including systemic lupus erythematosus, rheumatoid arthritis, ankylosing spondylitis, osteoarthritis, amyotrophic lateral sclerosis, multiple sclerosis, Alzheimer's disease, and fibromyalgia. The reverse is also true. Many cases of Lyme Disease have been found masquerading as rheumatologic ailments.(1,4) The manifestations of Lyme Disease have been outlined in three clinical phases: early localized, early disseminated and late disease. Early localized disease tends to occur within the first few days up to a month after the tick bite. It is characterized by Erythema Chronicum Migrans Rash (ECM) in 50-70% of patients, fatigue, malaise, lethargy, headache, myalgia, arthralgias, and regional generalized lymphadenopathy.

    Erythema chronicum migrans is classically described as a bulls-eye pattern with a red ring and a central area of clearing. The rash usually occurs around the site where the tick became imbedded in the host, but it may also appear at any site on the body and be totally unrelated to the site of attachment. In addition, the E.C.M. may appear as multiple ring-like rashes that intersect or remain distinct. Erythema chronicum migrans (which is pathognomonic for Lyme disease) enlarges over several days time and it represents the actual migration of spirochetes in the skin with an associated immune inflammatory response or erythema.

    Early disseminated disease usually occurs within several days up to 10 months after the tick bite. It includes symptoms of carditis in 8-10%, including conduction defects with bradycardia and mild cardiomyopathy, neurologic manifestations in approximately another 10%, including meningitis, encephalitis, cranial neuropathy - most commonly Bells palsy, peripheral neuropathy and myelitis.

    Musculoskeletal symptoms develop in 50% of untreated patients. Migratory polyarthritis and fibromyalgia, and other symptoms which might include lymphadenopathy, conjunctivitis, liver abnormalities, including hepatitis, and kidney abnormalities (including proteinuria) are common. About 50% of patients with early disseminated disease may develop skin rashes with multiple ring-like lesions.(10,11,12,13) Late disease commonly occurs months to years after the tick bite, and includes musculoskeletal manifestations. Approximately 50% of these patients develop migratory polyarthritis, and an additional 10% develop chronic monarthritis, usually in the knee; some may develop fibromyalgia. There are also neurologic manifestations which include subtle peripheral neuropathies, encephalopathies, ataxia, dementia, and sleep disorders. Some cutaneous problems may develop, as well.

    Making the diagnosis of active Lyme disease requires a history of one of the above clinical scenarios with supportive positive Lyme antibody serology (ELISA). Positive or borderline serology tests require confirmation by Western Blot analysis.

    Microbiology
    Three distinct species of spirochete have been isolated that are known to cause Lyme Disease in the United States, Asia and Europe: Borrelia burgdorferi, Borrelia afzelii, and Borrelia garinii. In addition, there are over 100 strains of the spirochete in the U.S. and 300 identified Worldwide. There are three species of ixodes ticks in the United States and Asia that are capable of spreading the spirochete: Ixodes scapularis (formerly Dammini - Eastern and North Central U.S.), Ixodes pacificus (Western U.S.), and Ixodes rincus (Europe). The life cycle of the tick is as follows:

    1. The ixodes tick larva hatch from eggs in the summer and search for a blood meal. They typically feed on mice (which harbor the spirochete reservoir). The larvae are not responsible for Lyme Disease since they must first feed on an infected mouse before they can become infected. Once infected, they may harbor the borrelia spirochete in their digestive tracts.

    2. The following Spring, after molting, the tick emerges as a nymph. The nymph searches for another blood meal and, in the process of feeding, the spirochete migrates from the digestive tract to the salivary glands of the tick where it may be passed on to the host. The greatest incidence of infection from the nymph is in the late spring, summer and early fall, since these are the times when the nymph is searching for a blood meal.

    3. The nymph drops off the host and molts into an adult which then feeds on another blood meal in the late fall, winter or early the following spring. The total life cycle spans about two years.

    Ticks often take up to 24 hours on a host to find a suitable site of attachment. It is estimated that an additional 24-36 hours is required to transmit the spirochete from a tick into a susceptible host. In one study, only about 1% of tick bites resulted in actual infection.(14) Fifty to sixty five percent of adult ixodes ticks have been found to be infected with the spirochete, while only 2030% of nymphs are infected in endemic areas.(4) Despite this different incidence of infection, it is the nymph that has been implicated in 90% of human infections. Even though a greater percentage of adult ticks are infected carriers of the spirochete, they are outnumbered ten to one by nymphs, which are also harder to detect. This is the reason why nymphs are far more likely to spread disease than the larger, more commonly infected adult.(1)

    Conventional antibiotic treatment is recommended in documented cases of Lyme disease and prophylactically if certain criteria are met. Antibiotic treatment is recommended with:

    1. Diagnosis of E.C.M. in an endemic region. There is no need for serologic confirmation before initiation of treatment in these cases.

    2. Diagnosis of active Lyme disease via serologic testing, when active symptoms are present. This requires a positive or borderline positive Lyme titer (ELISA) with confirmation by Western Blot.

    3. Recent history of a deer tick bite with attachment greater than 24 hours with or without confirmatory symptoms in an endemic area. NOTE: A positive serological test, without symptoms of active Lyme disease is not an indication for treatment. There is a 7% false-positive rate in the serologic (ELISA) test. Other illnesses can also produce false positive test results, including syphilis, endocarditis, Epstein Barr Virus, mononucleosis, rheumatoid arthritis, juvenile rheumatoid arthritis, systemic lupus erythematosis, pinta, yaws, bejel, leptospirosis, and malaria.(1)

    Generally, oral amoxicillin is considered first line treatment for children less than 9 years old, and oral doxycycline is first line treatment for adults. Initial treatment typically extends for 3-4 weeks.

    Co-infection with the disease-forming agents of Babesiosis and Ehrlichiosis may complicate the diagnosis and treatment of Lyme disease. Additionally, there are other tick-borne infections that may be confused with Lyme disease. Knowledge of all of these agents is necessary in making a thorough evaluation in a case of suspected Lyme disease.

    Ehrlichiosis: Human Monocytic Ehrlichiosis (HME) is caused by the organism Ehrlichia chaffeensis and is transmitted primarily by the lone star tick (Amblyomma americanum), but can also be transmitted by the American dog tick (Dermacentor variabilis). Whereas, the organism that causes Human Granulocytic Ehrlichiosis (HGE) has not yet been identified, but is known to be carried by the same tick that carries Lyme disease and Babesiosis (Ixodes Scapularis). Symptoms of HME or HGE include an acute febrile illness accompanied by headache, malaise, myalgia, fatigue, vomiting, anemia and rigors. Symptoms may mimic the flu, viral hepatitis, aseptic meningitis, pneumonia and cholecystitis. Less common symptoms may include cough, sore throat, diarrhea, lymphadenopathy, rash, seizures, abdominal pain and confusion. Untreated, mortality rate is up to 5% for HME, and about 7-10% for HGE. Diagnosis of Ehrlichiosis is made by examination of the blood from a peripheral smear. Since the Ehrlichial organisms are obligate intracellular parasites, they cannot be easily detected with the usual methods. Diagnosis currently depends upon direct visualization of the characteristic inclusion bodies in the white blood cells (monocytic or granulocytic) from an infected host.(15,16)

    Babesiosis: Babesiosis is caused by the protozoan Babesia microti and several related species. Like Lyme disease, it is also transmitted by the ixodes tick. Babesiosis may cause a febrile hemolytic anemia with potential for liver damage and renal failure. Symptoms include fever, chills, fatigue, headache, muscle pain, anemia, nausea, shaking chills and night sweats. In cases of Lyme disease that appear particularly severe, or if there is evidence of atypical signs and symptoms (including severe anemia, enlarged spleen, thrombocytopenia, or elevated liver function tests), then the diagnosis of co-infection with either Babesiosis or Ehrlichiosis must be considered. Co-infection rates run about 10-12% in endemic areas.(13,17,18) The diagnosis of Babesiosis is made by having a high clinical index of suspicion and is confirmed by examination of the peripheral smear which reveals intraerythrocytic organisms (which appear similar to Falciparum malaria). The conventional medical treatment of Ehrlichiosis is doxycycline or chloramphenicol. The standard treatment regimen for Babesiosis is clindamycin and quinine.(18)

    Rocky Mountain Spotted Fever: RMSF is caused by the organism Rickettsia rickettsii and is spread by the American dog tick (Dermacentor variabilis) and the wood tick (Dermacentor andersoni). Rock Mountain Spotted Fever has a 25% mortality rate in untreated cases. Early symptoms may include fever, rash, nausea, vomiting and cough. About 20% never even develop the well-known measles-like rash that starts on the extremities and spreads over the entire body. Advanced disease may include seizures and pulmonary edema. Diagnosis of RMSF is made by examination of the peripheral blood smear since the rickettsial organism is also an obligate intracellular parasite. Conventional medical treatment includes use of doxycycline or chloramphenicol.

    Colorado Tick Fever is a viral illness transmitted by the Rocky Mountain Wood tick. It is characterized by high fevers, chills, severe headache, muscle aches and occasional rashes. These symptoms may recur weekly, are usually self-limited and do not require antibiotic therapy.

    Relapsing Fever is caused by the Borrelia hermsii, Borrelia turicatae and Borrelia parker spirochetes. It is transmitted by soft ticks, mostly in the Western U.S . It is characterized by repeated episodes of fever, chills, headaches, muscle and joint pains lasting approximately one week. Conventional medical treatment includes penicillin or doxycycline.

    Tick Paralysis is a potentially fatal reaction to the toxin secreted by the feeding American dog and Rocky Mountain Wood Ticks. Symptoms include headache, vomiting and malaise, followed by an ascending paralysis that may progress to respiratory failure and death if untreated. Treatment includes removal of the tick from its site of attachment.

    Tularemia is caused by the bacteria Francisella tularensis and can be transmitted by the American dog, Lone Star, Rocky Mountain and Pacific Coast ticks. It may also be transmitted by horseflies, deerflies and contact with infected animals, including rabbits. Symptoms include recurrent fevers, generalized lymphadenopathy leading to ulceration, conjunctivitis and pneumonia. Conventional medical treatment includes streptomycin or tetracycline.

    Homeopathic Treatment
    The starting point for homeopathic medical treatment of Lyme disease and other related zoonotic infections is the history and physical exam. It is important to remember that the diagnosis of Lyme disease does not automatically imply the treatment, homeopathically. The homeopathic repertory is helpful after a case has been thoroughly perceived.

    Hahnemann stated in paragraph 6 of The Organon:
    The unprejudiced observer perceives nothing in each single case of disease other than the alterations in the condition of the body and soul, disease signs, befallments, symptoms, which are outwardly discernable through the senses. That is, the unprejudiced observer only perceives the deviations from the former healthy state of the now sick patient....All these perceptible signs represent the disease in its entire extent, that is, together they form the true and only conceivable gestalt of the disease.(19)

    A search through Murphy's Repertory (20) reveals three medicines under Lyme disease: Arsenicum album, Mercurius and Thuja. Expanding the search through the MacRepertory and ReferenceWorks programs, reveals four additional medicines: Carcinosin, Lac caninum, Ledum, and Syphilinum. I also include Tick Bite, Lyme Tick and Borrelia when evaluating cases of known or suspected Lyme disease or prophylaxis. These medicines are only a starting point for consideration. Any homeopathic medicine can be used to effectively treat Lyme disease if it is the simillimum to the individual case.

    In my practice, cases of prophylaxis following a tick bite receive a thorough constitutional analysis in addition to consideration of the above medicines and nosodes. When working with documented cases of newly diagnosed or chronic Lyme disease, a full analysis and repertorization must be completed prior to consideration of any medicines. The manner of this analysis follows the same guidelines as all cases treated from a classical homeopathic perspective:

    I. A complete history and a focused physical exam should always be performed.

    II. The diagnosis of Lyme disease is given equal consideration as an additional factor affecting an individual's health, but it is never taken as the total representation of the disease process unless there are no other signs, symptoms or significant history. In other words, the diagnosis of Lyme disease must be integrated into the total analysis of symptoms and considered as an additional factor affecting an individual's health. It must be considered a part of an individual patient's total pattern of susceptibility, referred to in paragraph 18 of The Organon where Hahnemann states:
    It is an undeniable truth that nothing can, by any means, be discovered in diseases whereby they could express their need for aid, besides the totality of symptoms, with consideration for the accompanying circumstances. Therefore, it follows incontestably that the complex of all the symptoms and circumstances perceived in each individual case of disease must be the only indicator, the only reference in choosing a remedy.

    III. Lyme disease must be considered in the context of the individual host who has proven to be susceptible. Illnesses manifest according to the patterns and cycles already in place in each particular individual. Through the judicious use of homeopathic medicines, according to the Law of Similars, an individual host's immune system may be augmented to provide the appropriate disease/ailment specific impetus that will enable effective, prompt resolution and re-establishment of equilibrium. Using homeopathic medicines, an individual's response to parasitic infestation may be directly augmented in a manner that provides safe, permanent resolution with minimal risk of resistance or recurrence.

    CASES
    1. Case one: A fourteen year-old prepubertal girl was brought to me for evaluation by her parents. They had noticed a ring-like lesion on her right thigh for the past week following what they thought was an insect bite about 10 days before. She complained of some pain in the thigh, but there were no systemic symptoms of fever, chills or viral-like illnesses. Emotionally she was described as excitable, high strung, and low key at home with an even temper. She reported occasional headaches both at home and at school, as well as occasional stomach aches. Her sleep was described as fine unless the weather was hot, and then she had difficulty since there was no air conditioning in the home. She slept on her side and occasionally propped her head up. She bruised easily. Acne was just beginning to manifest. Her diet was filled with vegetables, salads and grains. She was described as an avid reader of mysteries and a bright 8th grade student. No allergies, no medications.

    She had a history of mild lactose intolerance. She had braces on her teeth. She craved potato chips and lemonade. She was averse to asparagus and chili. There was a family history of colon cancer, headaches and allergies. She was not up to date on her immunizations and her family did not plan on completing them. There was a history of meconium at birth.

    Physical exam revealed: some mild acneiform eruptions on the face, and mild scoliosis; Tanner stage I breast development; normal pulmonary and cardiac exam and a clear bull's-eye rash on the left posterior thigh reaching around to the anterior thigh and inguinal area measuring 20 cm. in diameter. Neurologic and lymphatic examination was normal. Vital signs were normal and she was afebrile.

    My impression was that she displayed clear characteristics of an E.C.M. rash although there was no sign, yet, of systemic involvement. Her case history was imperfect, and, although I tried, I was unable to satisfy my appeal for a constitutional solution that matched her personality. Therefore she received a single dose of Lyme Tick 30C repeated in 24 and 48 hours for a total of three doses.


    My plan was to address a constitutional prescription at her follow-up in one month, but by that time she was completely free of symptoms. The E.C.M. rash had faded after one week. She had not advanced to develop any systemic signs of Lyme disease. She remains symptom free after eight months' follow-up.

    The medicine, Lyme Tick was prepared by me from a nymph stage deer tick removed from the neck of another patient who was successfully treated one year before. The tick was potentized to 30C and is now used as an isopathic nosode in certain cases of prophylaxis if a constitutional medicine is not apparent.

    Cases of Lyme disease prophylaxis pose a particularly interesting problem. The homeopathic medicine, Ledum, has been recommended by several homeopathic physicians for use in Lyme prophylaxis after a tick bite. This knee jerk prescription is reminiscent of the allopathic use of immunizations. To truly employ classical homeopathic theory according to the Law of Similars is extremely difficult in cases of prophylaxis unless there are strong symptoms or if the case is already known from prior experience.

    Several possible solutions present themselves in the dilemma of prophylaxis, including the use of homeopathic nosodes or the use of a constitutional prescription. Constitutional prescribing utilizes the complete analysis of an individual's total state of physical, mental and emotional characteristics that is present before Lyme disease manifests. Neither of these approaches are infallible. Professional judgment on each individual case might prove more beneficial.

    2 . Case Two: A 9 year-old female came to me in July 1996 with a tentative diagnosis of juvenile rheumatoid arthritis (JRA). The first episode had occurred in January of 1996, about a month after a deer tick had been found imbedded in her scalp. Her primary physician, at that time, did not believe that ticks could spread Lyme disease during the winter months and, so, he had treated her for JRA. Her symptoms included three episodes of bilateral knee swelling, leg swelling and a rash; also pain in the left elbow and left ankle. Her skin felt tingly and was noted to be hot and occasionally red around the affected joints. She reported feeling tired in the afternoons after about 1:00 p.m. Pain was present in the legs and had been there for the past three weeks. She would get crabby and restless and have to limp because of the pain in her knees. Her joints were better from external heat, better from movement, and she preferred being outdoors in the open air. She had a poor appetite and was chronically constipated. Her left knee was worse than the right, and it was worse from flexing. Her first episode (seven months earlier) had been marked by a fever, but she had not had any febrile response since that time.

    Her physical exam revealed a well-developed, well-nourished white female in no apparent distress. Her skin was fair. She had blond hair. Her temperature was 97.6. Her pulse was 80. Examination of head and neck was remarkable for shoddy cervical adenopathy. Cardiac and abdominal exam was within normal limits. The extremities were remarkable for a grossly deformed left knee with positive ballotment of the patella, with marked effusion. The knee was hot, but it was nonerythematous. Range of motion was limited due to pain. There were no skin rashes and no other lymphadenopathy was noted.

    My impression was that early disseminated Lyme disease had produced a monarticular arthritis with recurrent episodes of polyarticular arthritis since January 1996, following the tick bite. Other considerations included JRA and other rheumatologic conditions, as well as septic arthritis. The plan at that time included a Lyme titer with a complete blood count and erythrocyte sedimentation rate. I treated her with the medication Veratrum viride 200C. She took it once and then she plussed it every 24 hours, for several days.

    In one week's time, the Lyme titer was reported as being strongly positive, the ESR was elevated at 44 (normal 10-20), and her CBC was normal. At two week's follow-up, she felt much better; there was no pain, and only slight swelling remained in the left knee. She had been feeling that everything was steadily improving. She was instructed to stop the Veratrum viride and was given a single dose of the medicine Syphilinum 1M.

    She responded rapidly, and by her next appointment, three-and-one-half months later, she had not had any recurrences. She was absolutely free of joint problems and only one mild upper-respiratory infection had punctuated the interim. Her physical exam was completely normal, her cervical adenopathy had resolved, and her knees were normal. Since both JRA and Lyme are capable of producing a positive ELISA test, the definitive study at this point might have been the Western Blot. In the interest of saving cost, that test was deferred with the caveat that it would be ordered at the earliest sign of recurrent symptoms. She remains symptom free to this date without evidence of recurrence.

    Veratrum viride is American Hellebore. It was selected on the basis of the following rubrics from Murphy's Repertory (20):
    Ankles; SWELLING
    Environment; AIR, cold, agg.
    Generals; WEAKNESS; general,; afternoon; 1 p.m.
    Generals; WEAKNESS; general
    Joints; SWELLING, of
    Joints; DISCOLORATION, redness
    Knees; SWELLING; general; rheumatic
    Knees; SWELLING; general; hot
    Knees; RHEUMATIC, pain
    Knees; ACHING, pain
    Knees; PAIN, knees; general; turning, in bed; on turning the limb
    Knees; PAIN, knees
    Legs; ACHING, pain; lower
    Limbs; TINGLING, prickling, asleep
    Mind; QUARRELSOME
    Mind; COMPLAINING
    Muscles; PAIN, muscles,; general; rheumatism, in acute Muscles; PAIN, muscles,
    Neck; SUBMAXILLARY, glands; inflammation
    Neck; PAIN, glands; sides
    Syphilinum was administered in follow up on the basis of the following rubrics:
    Ankles; PAIN, ankles; rheumatic
    Generals; CHRONICITY, of complaints
    Generals; POSITION, change of body, agg.
    Generals; WEAKNESS; general
    Generals; MOTION, general; amel.; continued
    Generals; HOT, applications, amel.
    Knees; ACHING, pain
    Skin; RASH
    Knees; PAIN, knees
    Legs; PAIN, legs; lower
    Legs; ACHING, pain; lower
    Legs; SWELLING; lower
    Legs; ACHING, pain; lower
    Limbs; ACHING, pain
    Limbs; RHEUMATIC, pain
    Mind; DISCONTENTED, displeased, dissatisfied
    Mind; IRRITABILITY
    Neck; SWELLING of; glands
    Rectum; CONSTIPATION, general,; obstinate; years, for

    3. Case Three: A seventy year-old housewife with a history of Lyme disease diagnosed 7 years ago. She was treated, at that time, with oral doxycycline for 3 months, followed by several other antibiotics (including cefixime) for a total of 2 years of antibiotic treatment. When she came to see me, she said she continued to have myalgias, with aching in all the muscles in the body and, particularly, the back and across her shoulders. All her symptoms were worse in the morning and at night. The aches were present for 7 years and she had recently been diagnosed as having fibromyalgia. She said the pain would move in cycles, lasting about 11/2 weeks, and the pain would make her feel quite depressed. (Her primary physician had offered to treat her with antidepressants.) All her symptoms were worse in the cold, better in hot weather, better from warmth, better from motion, worse sitting still. She was occasionally constipated, worse from missing meals, and better from hot meals. She complained of an inability to think straight. She easily lost her train of thought, and had a history of orthostatic vertigo. She also had difficulty remembering names, frequent occipital headaches, dry eyes, cataracts, sciatic pain down both legs, and a history of hemorrhoids. She described a cold sensation in her legs, with tingling and burning when waking early in the morning. She complained of generalized pruritus, mostly on her back, and she had occasional chills. Past medical history was significant for a cardiac arrest in 1992 and hypertension, treated allopathically for 19 years. She also had a childhood history of severe Scarlet Fever where she was hospitalized, and her mother had told her that she almost died because of it. Her surgical history included hysterectomy, appendectomy, and a cholecystectomy. There was a family history of diabetes and depression.

    Physical exam revealed a well developed, well-nourished white-haired elderly woman; blood pressure was 180/98; pulse was 88; head and neck exam demonstrated left frontal and maxillary sinus tenderness, multiple dental amalgams and partial plates. Otherwise, her physical exam was normal.

    My clinical impression was poorly controlled hypertension (of note, she had recently discontinued her anti-hypertensive medications without the knowledge of her physician) and a long-standing history of Lyme disease (treated ineffectively with multiple courses of antibiotics) now in a late phase, mistakenly diagnosed as fibromyalgia and depression.

    She received the nosode Scarlatina 200 C as a single dose. At follow-up, three weeks later, she returned with complaints of a severe headache at the vertex and temples. She had also developed occasional chills and a post-nasal drip. Her body aches were significantly improved, and she had read three books (a definite sign of improvement since she was unable to concentrate long enough to finish a single page at her initial visit). Her physical exam was essentially unchanged and her blood pressure was 180/80. She was given Glonoine 200C as a single dose, and at her next scheduled follow-up 2 months later, she explained that she had continued to improve until she became nearly pain-free for the first time in 7 years.

    Scarlatina is a nosode prepared from the lysate from the squams of a patient with scarlatina. Scarlet fever is a little-known condition today associated with a Group A streptococcal pharyngitis. There is usually a diffuse cutaneous rash associated with the erythrogenic toxin. Rare cases of scarlet fever have been associated with pulmonary, hepatic and arthritic involvement. Little is known of the scarlatina nosode and (to my knowledge) it has never received a thorough proving. Clinical indications relate to the actual disease state. It has also been likened to the proving of Streptococcinum (which is available). Clinical indications for Scarlatina include weakness, headaches, vomiting, cervical adenopathy, nephritis, psoriasis, arthralgias and rheumatism of fingers and hands, chronic polyarthritis, and abdominal pains.


    Glonoinum was selected to follow Scarlatina on the basis of the following rubrics:


    Arms; TINGLING, prickling, asleep; general; side, lain on
    Arms; SHARP, pain
    Arms; ACHING, pain
    Back; PAIN, thoracic; middle of
    Back; COLDNESS; general; extending, down back
    Blood; HYPERTENSION, high blood pressure; general; sudden rise of
    Generals; PAINS; general; appear gradually; and disappear gradually
    Generals; SENSATIONS, general; tingling
    Generals; WEAKNESS; general
    Generals; WALKING, general; amel.
    Generals; MOTION, general; amel.; slow
    Head; HEADACHES; occiput; hammering
    Legs; PAIN, legs; lower
    Mind; MEMORY, general; forgetful; words while speaking, of, word hunting Mind; CONCENTRATION, general; difficult
    Mind; CONFUSION, of mind; talking, while
    Mind; CONFUSION, of mind
    Mind; EXHAUSTION, of mind
    Mind; MEMORY, general; forgetful
    Mind; MEMORY, general; weakness of
    Mind; MEMORY, general; weakness of; names, for proper
    Mind; THOUGHTS, general; wandering
    Muscles; SHARP, pain
    Rectum; CONSTIPATION, general,; hemorrhoids, from
    Shoulders; PAIN, shoulders

    The thread that runs between these cases and, particularly, the last two, is the notion that homeopathic medicine may be used effectively in the treatment of Lyme disease if there exists a constitutional similarity in the symptom picture produced by the medicine and the particular manifestations of the disease process in a particular individual host.

    DISCUSSION

    Rx: Homeopathic vs. Allopathic?

    It is important to recognize the clinical manifestations of Lyme disease and other zoonotic illnesses, and to understand the treatment options, using both the conventional and the classical homeopathic approach. The use of conventional antibiotic medicine conforms to the standard of care in this country, but even when implemented according to current treatment guidelines, does not guarantee that these illnesses will be cured. The number of cases of recurrent and chronic illness are increasing as the diagnosis of Lyme disease is more frequently made. In addition, there are many reported cases of post-Lyme disease syndrome and a vast number of individuals who find it difficult to fit into any diagnostic category and so are classified as chronic fatigue, fibromyalgia, somatization disorder or depression.(2)


    Antibiotics are not tremendously effective in treating Lyme disease at any stage. Initial treatment recommendations start with courses of therapy ranging 3-4 weeks followed by reevaluation, and then an additional course of several weeks or months if symptoms persist or recur. Late stage illness requires long courses of intravenous antibiotic therapy. Treatment is prolonged because these tick-borne organisms are relatively resistant to therapy, even at the outset. In addition, the longer any antibiotic therapy continues the greater the likelihood that resistance will develop. This is a hard lesson which we have already learned by experience with tuberculosis and other infections.


    The phenomenon of antibiotic resistance with subsequent recurrent infection, chronic infection and resistant strains of organisms has been extensively investigated and many recommendations made.(5,6,7,8,9) The blame for this phenomenon has generally been placed upon a combination of physicians' indiscriminate overuse of these agents, coupled with patients' demands and erratic compliance with their proper use. This tendency to blame the physician (for trying to eradicate an illness quickly and efficiently) or the patient (to be free of illness rapidly and to stop taking a potentially toxic medicine when they feel better) may be a mistake based on a basic incorrect assumption and a false premise about how antibiotics work in treating illnesses.


    Antibiotics do not cure infections. This is a common misconception, even among physicians. When antibiotics are used judiciously in cases of susceptible bacterial infections in concentrations that are considered bactericidal, then a proportion of the total bacterial load is reduced in a dose-response relationship. Through repeat dosing over days, weeks or months, bacterial counts are significantly reduced so that the host's immune system can (theoretically) complete the task of eradicating any remaining organisms. The task of healing, or reestablishing homeostasis is always dependent upon a number of different factors intrinsic and extrinsic to an individual, but includes the orchestration and coordination of the entire organism; mental, physical and spiritual.(21)


    The pharmacodynamics of antibiotic use necessitates a level toxic to the bacterium be established within a range that is tolerable to the host. In The Pharmacological Basis of Therapeutics, Goodman & Gilman state:


    The dose of a drug utilized must be sufficient to produce the necessary effect on the microorganisms; however, concentrations of the agent in plasma and tissues must remain below those that are toxic to human cells. If this can be achieved, the microorganism is said to be susceptible to the antibiotic. If the concentration of drug required to inhibit or kill the organism is greater than the concentration that can safely be achieved, the microorganism is considered to be resistant to the antibiotic.

    As this bacteriocidal range is approached, the rate of bacterial inhibition or toxicity approaches a level of efficacy, but (by nature of biological systems and pharmacodynamics) becomes asymptotic to the maximal expected efficacy. This means that even the best antibacterial agents are never 100% effective in eliminating any parasitic organisms, except in ideal in vitro conditions where toxicity to the host is not an issue.

    If antibiotics are not responsible for curing bacterial infections, how is it that the majority of individuals treated with these drugs seem to improve? Clearly, when we improve from any condition, including infectious diseases, it is a result of the body's own tendency toward homeostasis (working through the immune and other systems) that enables a cure. Goodman and Gilman continue:


    An important determinant of the therapeutic effectiveness of antimicrobial agents is the functional state of the hosts defense mechanisms. Both humoral and cellular immunity are important. Inadequacy of type, quality, and quantity of the immunoglobulins, alteration of the cellular immune system, or either a qualitative or, most important, a quantitative defect in phagocytic cells may result in therapeutic failure despite the use of otherwise-appropriate and effective drugs.(22)

    The apparent eradication of infectious organisms may actually be a partial shut down of the immune/inflammatory response that provides the symptoms of infection. In other words, if the antigenicity of the organism (infectious agent) can be changed or modified to create less of an immune response (see below), or if our ability to react immunogenically to the organism has been suppressed, we will not be aware of the illness; we will have no symptoms. The organism would then be free to enter a less obvious, yet persistently active resident state resulting in damage in deeper structures over greater periods of time.

    At the microbiologic level, one of the ways that health is reestablished is through the coordination of all the arms of the immune system. Antibiotics and other agents that are utilized in assisting in immune function work at a very superficial and, perhaps, harmful level. Chemotherapeutic agents act by lowering the burden of parasitic opportunists and simultaneously turning off the autonomic inflammatory immune response. These chemotherapeutic antibiotic agents may not only be somewhat incidental to actual cure; they may also act by effectively thwarting the body's own mechanisms of defense. Macroscopically, the use of these agents effectively turns off defense mechanisms of inflammation and fever (which are helpful in the immune response). These agents may interfere at the microscopic level as well.

    Goodman and Gilman further state that:
    Another interesting twist that may influence the efficacy of antimicrobial therapy is that these agents have been shown to affect various host immune responses adversely; these include leukocyte chemotaxis, lymphocyte and monocyte transformation, antibody production, phagocytosis, and the microbicidal action of polymorphonuclear leukocytes. While the clinical significance of this immunosuppression is not known, these observations should help discourage the indiscriminate use of antibiotics.(22)

    We have no studies demonstrating that a sterile environment is ever achieved internally. All evidence points to the fact that we are constantly surrounded (internally and externally) by potentially pathogenic organisms. It is only through the constant activity of our immune systems, supported via physical, emotional and spiritual means, that we maintain health. Healing, when it does occur, is ultimately the result of host factors that continue to succeed in mounting surveillance coupled with a lytic response. Symptoms act as essential feedback loops maintaining this dynamic equilibrium while triggering appropriate biochemical, emotional and behavioral responses.

    As a result of even the most prolonged course of antibiotic treatment, some bacterial organisms do survive within us. Those that do survive, by definition, have acquired a degree of antibiotic resistance. Therefore, the very nature of antibiotic therapy favors the Natural Selection of more resistant infectious agents that are able to exist and survive without immune inflammatory recognition. Conventional antibiotic therapy directly promotes this course of events.

    Another result of antimicrobial therapy complicates diagnosis. As the immune response is shut off (prematurely by reduced bacterial count and direct inhibition), antibody production and cellular memory may become impaired leading to a delay or an inhibition of seroconversion. This would effectively reduce the reliability of antibody-based diagnostic tests.(23)

    The very basis for the germ warfare approach to infectious illness enables and ensures the retaliation from stronger bacterial and viral organisms in future illnesses. It should be no surprise that even through the most judicious use of these agents, coupled with the most thorough (and compliant) courses of treatment, that emergence of resistance to these agents is a guaranteed ultimate fact. Recurrence is only a matter of time and (in a dynamic living system) a question of location.

    From this discussion, it would appear that our immune system has developed through a reliance upon the growth characteristics of infectious organisms within and around us. The orchestration of antigenic recognition, clonal production and cellular memory may not have sufficient information to adapt to the changes that we so readily interject. The use of chemotherapeutic antibiotics (that reduce, but fail to eliminate these organisms) may exacerbate the situation and hinder the immune response by confusing feedback loops of inflammation. If antibiotics are capable of interfering with our system in this manner, they may shut down important processes prematurely. This may directly lead to a form of revolving door series of infections until suppression was strong enough to set up a chronic illness (see below). The strategy at the core of the conventional approach to infectious disease may be fundamentally flawed. The notion of a sterile (bacteria free) human is as absurd as the notion of a perfect vacuum - it does not exist. Additionally, our ignorance in relying on this Stone Age view of microbiological dynamics may be extremely harmful. Our attempts to assist with infections using antibiotics may significantly impede our health.

    If we recognize that antibiotic resistance is an inevitable fact, then we will also perceive that the phenomenon of disease suppression is the ultimate outcome. Disease suppression is the phenomenon widely recognized for over 200 years by homeopathic physicians; it is one of their greatest contentions with allopaths. In this paradigm, the allopathic application of medicines (that only achieve the short-term reversal of symptoms) drives illnesses deeper into potentially more serious levels of pathology. This phenomenon (known only empirically till now) has recently been validated.

    Suppression is seen following many conventional treatments using hormones, steroids, antibiotics and other medications that limit the inflammatory response. The use of these agents sometimes results in dire infectious, neoplastic, rheumatologic and metabolic consequences. Recently, the organism Chlamydia pneumoniae has been implicated in the pathogenesis of both heart disease and cerebrovascular disease(24,25). The bacterium Helicobacter Pylori has been implicated in the development of peptic ulcer disease, migraine headaches(26) and cancer(27). Both of these organisms are common superficial opportunists that appear to have escaped immune detection until their pathogenicity is expressed in an heretofore unsuspected disease. These are examples of potentially lethal consequences that may occur when the normal homeostatic balance of the immune system is turned off and suppressed by conventional treatments. In a similar fashion, viral agents have long been implicated in the development of certain types of cancer and lymphoma and, recently, a Borna virus has been implicated in the etiology of certain emotional ailments, including depression.(28)

    The change in role of these minor infectious agents and their involvement in more serious pathology may have resulted from the overuse of suppressive treatments that (a) effectively enabled resistance, (b) simultaneously inhibited immune recognition allowing the organism to evade detection and reach a deeper protected niche in arterial walls, in the mucus barrier inside the stomach or even in the brain.

    Recent studies in the pathogenesis of viral factors in cancer have indicated that the EBV agent is able to selectively express only certain proteins that enable it to remain protected intracellularly for long periods of time before contributing to a devastating illness.(29) Similarly, any form of therapy that suppresses immune function and reduces symptoms of inflammation (without eliminating the cause) may open the door to this phenomenon.

    In the example above, of cardiac and cerebrovascular disease, these may be the result of treatment with suppressive agents that acted to relieve symptoms, but inhibited the development of immunity. Short-term effects of this might include recurrent infections while long-term results might include pathology in more vital organs in the viscera. The recognition of the possible relatedness of these events has been delayed because conventional wisdom ignores the theoretical (and empirical) conclusion that different illnesses suffered by an individual throughout his or her lifetime may be related. It is interesting that the denial and flagrant ignorance of these phenomena now lead directly to their proof.

    The solution to this modern dilemma is not to redouble the use of suppressive therapies, but to work to stimulate and support the immune system through our understanding of how it does work. The recognition that our very own chemotherapeutic agents inhibit our immune defenses while strengthening the position of the parasite should allow us to explore the use of alternative therapeutic modalities that work in accordance with the functioning of the body rather than against it.

    In the case of Lyme disease (and many other infectious illnesses), if the immune reaction is turned off prematurely (by reduction of bacterial burden, below a threshold level), before all the arms of the immune response are activated, then the complex system of antibody production and cellular memory may not become fully activated. This may lead to a partial crippling of the very system that we have so generously tried to assist. The ability of the immune system to adapt to infection may be thwarted and subclinical disease may progress unhindered. The heroic, short-term, symptomatic solution may be more harmful in the long-run. Immunity may never develop and the illness may progress to more serious stages of (initially) asymptomatic pathology. This situation may lead to the successful, temporary relief from a symptom at the expense of worsening overall health.

    Biologically, the long-term effects of this short-term disease suppression may produce a profound dependence upon the repeated administration of these toxic agents and a weakening of the health of an individual as illness is ultimately driven deeper toward more vital organs. This phenomena should be perfectly clear by observing the effects of repeated antibiotic administration in cases of recurrent pediatric otitis media and strep pharyngitis. Conventional medical wisdom assumed that there was a long term benefit to the pharmacologic control and temporary suppression of these symptoms. However, these assumptions have never been rigorously tested in any double-blinded format. One recent study does suggest that antibiotics should not be indicated as first line treatment in these infections and that they should be regarded as optional.(31) This is the conclusion that has been supported by over 200 years of homeopathic treatment.

    Hahnemann states in paragraph 60 of The Organon:
    When these ill-consequences arise from the antipathic employment of medicines (as may very naturally be expected) the ordinary physician believes he can aid his cause by giving, with each renewed aggravation, a stronger dose of the medicine. This results, likewise, in only a short-lasting pacification. Since this necessitates an ever higher intensification of the palliative, there ensures either another greater malady or frequently even incurability, danger to life or death itself, but never cure of a malady that is old or very old.(19)

    Homeopathic medicines have been shown to stimulate an individual's immune response with an information/energy intensive quantum boost. This specific boost acts in a fashion similar to the crude inoculation, but appears to be individually matched to the precise pattern and vibratory frequency of the system that is out of atunement. This impetus enables the development of an increased strength of the immune response with greater resistance to illness. The immune system appears to benefit via this paradoxical treatment. Ultimately, homeopathic treatment forces the immune system to work harder in exercising immunity and cellular memory. Through the judicious use of the classical homeopathic method whereby each case is taken individually and the simillimum is given in the correct strength, we begin to see illness prevented and cured. In this manner, many illnesses that have become chronic and long-standing can be fully reversed.

    CONCLUSION

    My clinical experience with Lyme disease strongly supports the role of classical homeopathy and homeopathic medicines early after exposure as the most effective means of management. Since this application of medicine stimulates and augments the immune inflammatory response, a prompt resolution usually results. In early cases and in prophylaxis, I advocate the use of homeopathy as a first line of treatment. This approach is effective for the prompt resolution of threatening infection, and it additionally appears to stimulate the individual in a fashion (similar to an immunization) such that immunity from future infection is also strengthened (rather than the reverse, which is commonly seen with conventional antibiotic prophylaxis).

    My experience working with established cases of late Lyme disease has included cases treated with extensive courses of oral and IV antibiotic therapies for recurrent disease. At this stage, there is some controversy regarding the questions of chronic suppressed disease versus actual reinfection and true recurrence. The point is moot however since reinfection suggests that immunity was never attained on a cellular level. Antibiotic treatment suppressed not only the disease associated organism (spirochete), but also turned off the immune response prematurely and prevented the development of immunity.

    Chronic infection suggests that a stalemate of continued disease activity is the result of an associated disease agent (spirochete) that has established a niche in a susceptible host. Neither the antibiotic nor the host's immune response is sufficient to break the cycle. The continued use of stronger and broader spectrum agents only weakens the immune system further and allows the infection to slip deeper toward organ systems less able to evoke a perceptible symptomatic inflammatory response. It becomes more difficult to effect a cure using either homeopathic or allopathic medicines as the bacterial or viral agents become established in these deeper niches and symptoms subside. In this model, superficial infections may establish chronic silent residence until inevitable pathology ultimately effects more critical levels of functioning.

    Homeopathy is the most viable option in these cases as conventional treatments tend to act in a toxic manner toward the host, provide only partial bactericidal action and further suppress toward more serious latent illnesses. My experience suggests that the judicious use of homeopathic medicines applied through the classical homeopathic approach is one of the best means to cure Lyme disease (and other related infections) and to reestablish a healthy equilibrium.

    Acknowledgment

  2. grace54

    grace54 New Member

    I need to research homeopathic treatments more as conventional medicine hasn't helped me much. Thanks for the good article
  3. wrthster

    wrthster New Member

    You are very welcome! You would think there would be a lot more on here that would want this information. You try to help and no one reads it. I sure as well would give this a shot before ABX! A hell of a lot safer!
  4. wrthster

    wrthster New Member

    Your very welcome, and good luck to you!
  5. munch1958

    munch1958 Member

    I've tried homeopathy and found it to be very helpful. Remedies are very powerful and should only be used under the guidance of a practioner. Remedies sold over the counter are not as powerful as those used by a practioner. I wouldn't dabble with this on my own for Borrelia.

    This seems to be an older article. I do want to point out that Lyme is in every state most likely due to mosquitos and birds. Just because the CDC refused to recognize it that does not mean it's not there. People move around by going on V-A-C-A-T-I-O-N. They also R-E-L-O-C-A-T-E.

    To say that it's not in Colorado is misleading since there is Colorado tick fever or Rocky Mountain spotted fever. The vector in Colorado is Borrelia bissettii which has been isolated in mice found in the foothills.

    http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=87200

    Lyme disease in Montana is not recognized by the CDC because they have the wrong kind of ticks. They have Rocky Mountain Wood ticks.

    Source of article:
    http://www.canlyme.com/montanalyme.html

    Through the years, Lyme disease in Montana has been an open-and-shut case. To Montanans who thought they had it, the response from health officials has been the same: You couldn't.

    Montana has the wrong kind of ticks. It's deer ticks that carry Lyme disease, and Montana's ticks are Rocky Mountain wood ticks.

    "We've told people, 'We don't know what you have, but we know what you don't have, and that's Lyme disease,' " said state epidemiologist Todd Damrow.

    But Damrow and others were troubled by a nagging repetition of a similar set of symptoms reported by patients and by physicians: fever, severe fatigue and a bull's-eye rash at the site of a tick bite on the patient. A couple of years ago, Damrow got a photograph of a perfect bull's-eye rash from a public health worker in Montana. The rash was on a patient who had not left the state. At the same time, the Centers for Disease Control and Prevention were investigating a connection between Lone Star ticks in the Southeast and similar symptoms. They found a Lyme disease-like agent in the tick.

    "So I thought, 'Well, gosh, maybe I should not be so cavalier when we get these reports,' " Damrow said.

    The result was a study that began last tick season. The hypothesis is this: Could Montana have a similar phenomenon in its ticks, a Lyme disease-like agent that has adapted to the wood tick and is causing this similar illness?

    "We know we don't have Lyme disease, because we don't have the right kind of tick," said Pam Goldberg, an infectious disease specialist at the Missoula City-County Health Department. "But we might have a cousin to it."

    The study is a collaboration among local health departments around Montana, the state Department of Public Health and Human Services and the federal Rocky Mountain Laboratories in Hamilton.

    "We're real excited about it," Damrow said. "Doctors are concurring with this, and they think we're right on line."

    With western Montana's warmer weather in the past couple of weeks, ticks have appeared. Around Missoula, they've been picked up on Waterworks Hill, Mount Sentinel, Mount Jumbo and Blue Mountain. Hikers and bikers who extract blood-sucking ticks should think twice before crushing or flushing them.

    "When you get bitten by a tick," Damrow said, "don't throw it away because it may be of scientific value."

    Here's how it works: People who find ticks actively biting themselves or other people - not pets - should carefully remove it using tweezers or protected fingers. Pull steadily up and out; jerking may break off pieces of the tick's mouth under the skin, which can cause infection. Disinfect the bite and wash your hands. Put the tick in a plastic bag or jar, label it with your name and phone number, where it was found on the body and where it was picked up and take it to your local health department. Local health officials will send the ticks to the state.

    If you develop a bull's-eye rash around the tick bite, see your doctor. The doctor will draw a blood sample for state scientists to look at. Two months later, another blood sample will allow scientists at the state and at Rocky Mountain Labs to check for an antibody the patient may have developed to fight the illness. The health department will also want to take a photograph of the rash.

    Ticks that bit a person who developed a rash will be tested for evidence of infection at Rocky Mountain Labs, said Tom Schwan, a senior investigator and acting chief of its laboratory of human bacterial pathogenesis.

    "We'll try to detect the DNA from the bacteria that we suspect will be in them," Schwan said. "That could lead to an association between an infected tick and the symptoms in the human."

    The labs, which Schwan said are still waiting for official approval of the study from their parent agency, the National Institute of Allergy and Infectious Diseases, will use a test to amplify the DNA in the organism taken from the tick and map its sequence. Scientists will then compare that sequence to other organisms in a database, looking to prove the suspicion that illness is being caused by bacteria related to the Lyme disease spirochete.

    "We will either make that link or not make that link," Schwan said.

    If that link is solidified, then it can be said there's a public health risk, he said.

    With help from the University of Montana and UM professor Mike Minnick in the Department of Biological Sciences, the study's protocol has been approved by the Investigation Review Board, Damrow said.

    Another benefit of the study will be the ongoing look at the Lyme disease-like illnesses people develop. Because these cases haven't been followed, it's not known if they go on to the neurological and arthritic involvement characteristic of Lyme disease, Damrow said.

    "It's significant," he said, "because we really haven't characterized these cases."

    Last year, 345 ticks were collected around the state. Six of them were definitively linked to a photographed or physician-documented rash. For those six cases, paired blood samples were also collected.

    All this is not to say that people should try to attract tick bites for the advancement of science. Montana's own wood ticks can be infected with the bacteria that cause Rocky Mountain spotted fever. One or two cases occur in Montana every one or two years, Damrow said.

    "Untreated, it can be fatal," he said. "It is a potentially serious illness that is transmitted by our ticks here."

    The risk of infection is reduced if an embedded tick is found and removed before three hours have passed, Goldberg said.

    It's a good idea to check your dog, too, after you've been outdoors, she said. While ticks on dogs are not being studied, dogs can bring ticks into the house that can migrate to humans.

    The best defense outside is to keep covered with clothes that fit snugly around the wrists and ankles. Ticks wait on bushes and plants with their front legs ready to grasp anything that brushes against them. Right now, they are out in force.

    "The temperature's just right," Goldberg said. "It's warm. We have the sun. Right now it's the peak." Reporter Ginny Merriam can be reached at 523-5251 or at gmerriam@missoulian.com

    Lyme is not recognized in Missouri. It's called Master's Disease there.

    Source: http://www.canlyme.com/masters_disease_stari.html

    Master’s Disease:
    Named after a persistent physician who finally convinced the CDC that he was seeing numerous patients from Missouri and surrounding areas with Lyme-like symptoms, investigators discovered a “new” bacteria (Borrelia lonestari) carried by the Lone Star Tick which did not test positive using the normal Lyme disease tests. The rash caused by the bite is similar to that of Lyme disease, but sometimes raised and warm to the touch. Antibiotic treatment is the same.

    STARI – Southern Tick-Associated Rash Illness
    This may or may not be Master’s disease, but researchers are investigating an outbreak of tick-associated rashes in the southwest, again caused by the Lone Star tick.

    It is important to remember that any given tick may carry more than one infection, and that if you find one tick on you or a pet there may be 100 more “out there.”
    Tick Paralysis on the rise?

    Several cases of tick paralysis were recently reported in New Jersey. This disease is caused by the Dog (wood) tick, which secretes a neurotoxin as it feeds on the head – usually at the back of the neck. Initial symptoms may be slurred speech and weak legs, but the disease progresses to the point where the victim becomes paralyzed and has to be put on a ventilator. There is no treatment, but once the tick is found and properly removed symptoms resolve within 18-36 hours.

    Most susceptible are young children, especially girls, whose long hair may provide an ideal hiding spot for the tick, which will attach for a week or more to feed. As it does so it swells the size of a small olive, so don’t forget the daily tick checks.
    Tick-bits
    A question has arisen as to how ticks survive the winter months. Ixodes scapularis (deer tick) becomes an adult in the fall and actively searches for a host (usually a deer) from October to May whenever it is over 35-40°. The female attaches to its host and once mated fills up with blood, falls off and spends the winter “buried” in leaf litter. Eggs are usually laid in the spring, and the adult dies. During prolonged bouts of cold weather (snow is, to them, a quilt) they enter a phase called diapause, and become active again as it warms up. Ticks have been frozen in ice cubes for six months under laboratory conditions only to be walking around 24 hours later when thawed.
    Did you know that:
    *Part of the blame for the declining moose population in Maine has been attributed to the Winter Tick, which attaches to animals in such numbers that they can remove all the blood, causing the host to die. This tick can also kill deer, elk and other large mammals.

    *Unlike the Deer Tick, which waits for its prey, the Lone Star Tick is extremely aggressive and may travel 30-40 feet towards the source of carbon dioxide (humans) in search of a blood meal. It is not uncommon to find hundreds of these ticks on you in a very short period of time if you walk through or lie down in an infested area.

    *Tularemia, a bacterial disease which infects about 200 people per year in the United States, has been blamed for the death of a 17 month old toddler in Delaware. Often know as “rabbit fever” or “deer fly fever,” the disease is on the government’s “watch list” as a possible bioterrorist tool. In this instance the boy was bitten on the right ear, developed a fever of more than 105°, had swollen lymph nodes, muscle aches and weakness, and died several weeks later.
    Lyme Disease Vaccine Pulled From Market
    Citing poor sales, Lymerix, manufactured by GlaxoSmithKline, has been pulled from the market. In addition, an application to market a pediatric version has been withdrawn, and all Lyme disease research has been halted.
    New Tick-borne Diseases
    With support from the ALDF's Research Program, recent studies at Yale's School of Epidemiology and Public Health revealed a new spirochete resembling B. miyamotoi, which causes relapsing fever in Japan. Further studies indicated that the spirochete is found in 10-20% of ticks studies in New York, Connecticut, Rhode Island and New Jersey, and if implicated in human disease, may help explain late-stage symptoms exhibited by some people. In addition, the research indicates there may be other undisclosed microbes lurking within Ixodes scapularis.

    Only time will tell whether this new spirochete, as well as others yet to be "discovered," play a significant role in human disease.


    Rash not necessarily a "bull's eye"
    Results of a study recently reported in the Annals of Internal Medicine indicate that Erythema migrans often presents as a homogenous rash rather than a distinct "bull's eye," especially in early stages of Lyme disease. The rash is thought to occur in only 30% of victims.

    Data from the study indicates clinicians should change their approach to diagnosis.

    Timing of Blood Tests
    Time and again this office receives phone calls from patients who have a rash, and in some cases had had a tick attached at the site, only to be told by their physician to come back a month later for a test. It is imperative that clinicians understand that the presence of a homogenous or "bull's eye" rash caused by a tick-bite is indicative of infection, and treatment should be started immediately. Most blood tests do not work until 4-6 weeks after infection has occurred, and any delay may cause complications at a later date.



  6. kking0412

    kking0412 New Member

    will have to print that, too much to read right now.

    I just started flaring, I think? and am trying a homeopathic that has worked for a church friend of my mom's since 1999. It's called Ledum, and the protocol is kind of tricky (how do you remember how often to take stuff when you can't remember squat?) but it's 'Ledum Palustre' 1m. am on the first week and so far I can honestly say the joint symptoms have not gotten worse. of course I picked this week to trip and fall over the big black dog, so...

    Also: Therapeutic grade essential oils ~ I was told to use Idaho Tansy over the liver area for lyme.
  7. tansy

    tansy New Member

    Hi

    Good luck with your remedies; Ledum has helped others too.

    Homeopathy does not seem to work particularly well for me but when my son was young homeopathic remedies enabled us to manage his asthma without having to resort to oral steroids.

    TC, Tansy
    [This Message was Edited on 10/14/2007]
  8. lynne11

    lynne11 New Member

    I would like more info regarding homeopathics and Chronic Rocky Mountain Spotted Fever. I cannot find any doctors that believe that this is an actual issue. I have been reading the research of Dr. Cecile Jadin, she currently resides in South Africa.
    I was diaganosed, on my death bed, in June 2009, with Rickettsia. I now have issues that are almost unbelievable, except to those people that have been bitten by infected ticks.
    I just want to be well!!!
    Please share any available data!
    Thanks in advance!
  9. munch1958

    munch1958 Member

    Hi Lynne:

    Check out this link:

    http://www.pureformulas.com/rocky-mtn-spotted-fever-10-vial-kits-by-deseret-biologicals.html

    I've just completed the target therapy series for Borrelia, Babs & Ehrlichia (1 set) and the Bartonella series. I am almost done with the candida series; just two more vials to go.

    Candida:
    http://www.pureformulas.com/candida-albicans-10-vial-kits-by-deseret-biologicals.html

    Bart:
    http://www.pureformulas.com/bartonella-10-vial-kits-by-deseret-biologicals.html

    Bb, Babs, Ehrlichia:
    http://www.pureformulas.com/borrelia-10-vial-kits-by-deseret-biologicals.html

    Then will move on to the EBV series, then CPN, and mycoplasma and then hopefully I will be done with this crud. My goal is to do the series for all of my stealth pathogens.

    I don't care for the single formulas as much as I like the targeted series. There is a schedule in the box lid that I followed. Most of these were one vial every 3 - 7 days. I am completely off antibiotics, anti-fungals and antivirals now.

    I hope you have good results if you try this too!
    Linda