stomach problems

Discussion in 'Fibromyalgia Main Forum' started by Hawkeye, Mar 4, 2007.

  1. Hawkeye

    Hawkeye New Member


    Does anyone else experience severe stomach pain and gastritis? I have had this for years off and on and I am having a huge flare right now. I know I need to just go to the Dr but then they go right into refering to a GI who then wants to do endoscopy and other time consuming expensive tests which I do not have the money or time to endure right now.

    Does anyone know of any supplements or herbs that can help settle the intestine?

    I am desperate and tired of feeling stomach pain every single day.

  2. charlenef

    charlenef New Member


    dgl licorice root and guacatonga no side effects ive been taking them for about 8 months i can finally eat almost everything ill try and put some info about them for you charlen e
    you only need to read the section on the dgl licorice root all the bad stuff that raises your blood presser is taken out
    [This Message was Edited on 02/24/2007]



    [ edit | delete ] GUACATONGA 02/24/07 09:04 AM

    Family: Flacourtiaceae
    Genus: Casearia
    Species: sylvestris
    Synonyms: Samyda parviflora, Casearia parviflora, Anavinga samyda
    Common Names: guacatonga, guassatonga, wild coffee, burro-kaa, café-bravo, cafeiillo, café silvestre, congonhas-de-bugre, corta-lengua, crack-open, dondequiera, erva-de-bugre, erva de pontada, guayabillo, mahajo, papelite, pau de lagarto, piraquina, raton, sarnilla, ucho caspi
    Parts Used: Bark, Leaves

    From The Healing Power of Rainforest Herbs:

    Main Actions Other Actions Standard Dosage
    protects stomach
    blocks pain signals
    prevents ulcers
    neutralizes venom
    Infusion: 1/2 cup 2-3
    kills cancer cells
    kills viruses
    times daily
    slows tumor growth
    cleanses blood
    Capsules: 1-2 g twice daily
    relieves pain
    stops bleeding

    heals wounds


    Guacatonga grows as a shrub or small tree usually 2 or 3 meters tall, but sometimes grows up to 10 meters in undisturbed areas of the Amazon. In the clay soils of the Amazon, the plant has adapted for nutrient absorption and support by forming extensive lateral roots that are white, stiff, and covered with a corky bark. The tree produces small white, cream, or greenish flowers (which smell like a mixture of honey and urine) crowded on short stalks on the leaf axils. After flowering it produces small fruits, 3-4 mm in diameter, which split open to reveal three brown seeds covered with a red-to-orange aril. Guacatonga grows wild throughout the tropics, adapting to both forests and plains. It is native to Cuba, Jamaica, Hispaniola, Puerto Rico, the Caribbean, Central America, and South America (including Brazil, Peru, Argentina, Uruguay, and Bolivia).


    Guacatonga has a rich history in herbal medicine systems in nearly every tropical country where it grows. The Karajá Indians in Brazil prepare a bark maceration to treat diarrhea; the Shipibo-Conibo Indians of Peru use a decoction of the bark for diarrhea, chest colds and flu. Other Indian tribes in Brazil mash the roots or seeds of guacatonga to treat wounds and leprosy topically. Indigenous peoples throughout the Amazon rainforest have long used guacatonga as a snakebite remedy. A leaf decoction is brewed that is applied topically and also taken internally. The same jungle remedy is used topically for bee stings and other insect bites. This native use found its way out of the rainforest and into current herbal medicine practices in cities and villages in South America. It has been validated by scientists in the last several years who documented the leaf extract as capable of neutralizing several types of bee and snake venoms.

    Guacatonga has a long history of use in Brazilian herbal medicine, documented in early folk medicine books as an antiseptic and wound healer for skin diseases (in 1939), as a topical pain-reliever (in 1941), and as an anti-ulcer drug (in 1958). It is currently used in Brazilian herbal medicine systems as a blood purifier, anti-inflammatory, and antiviral to treat rheumatism, syphilis, herpes, stomach and skin ulcers, edema, fevers of all kinds, diarrhea, and as an topical pain-reliever. It is also employed topically for burns, wounds, rashes, and such skin disorders as eczema and . The natural herbal remedy calls for 20 grams of dried leaves infused in 1 liter of water; quarter-cup amounts are taken orally 2-3 times daily.

    The plant is also a popular herbal remedy employed in Bolivian herbal medicine, where it is considered to relieve pain, reduce inflammation, reduce stomach acid and prevent ulcers, stop bleeding and heal wounds. There it is used to treat skin diseases, cancer, stomach ulcers, snakebite and bee stings, herpes, and in dental antiseptic mouthwash products.


    The chemical makeup of guacatonga is quite complex. Scientists conducting the antivenin research discovered that the leaves and twigs of the plant contain a phytochemical called lapachol. This is the well known and studied anticancerous/antifungal compound from which another rainforest plant, pau d'arco (Tabebuia impetiginosa), gained much renown. (Pau d'arco is also featured in this book.) While other researchers have been studying the anticancerous and antitumorous properties of guacatonga, a completely different set of phytochemicals has fueled their interest. These compounds, called clerodane diterpenes, are found abundantly in guacatonga and some have been patented as antisarcomic agents. Clerodane diterpenes have been documented with a wide range of biological activities ranging from insect antifeedants, to antitumorous, anticancerous, and antibiotic agents, to HIV replication inhibitors. Some of the clerodane diterpenes documented in guacatonga are novel chemicals which scientists have named casearins (A thru S). Other chemicals in guacatonga include caprionic acid, casearia clerodane I thru VI, casearvestrin A thru C, hesperitin, lapachol, and vicenin.


    The research on guacatonga's anticancerous properties began in 1988 by Japanese researchers from the Tokyo College of Pharmacy and Pharmacognosy. They published one preliminary trial in 1988 on their discovery of these novel clerodane diterpenes and their anticancerous and antitumorous activities. The study indicated that an ethanol extract of the leaf showed strong antitumorous activity in laboratory mice with sarcomas. As soon as they made this discovery, they rushed to patent it, filing a Japanese patent for the casearin chemicals they'd discovered as new antitumorous agents. They published a follow-up study in 1990, again reporting their results from injecting mice with sarcomas with an ethanol extract of guacatonga leaves (100 mg per gram of body weight) and confirming their previous findings. They then tested individual casearins against various human cancer cell lines and published two more studies in 1991 and 1992. These studies reported newly isolated casearin chemicals and their antitumorous and anticancerous actions against various cancer tumor cells. Oddly, the Japanese researchers have not published any further studies and, since they had already filed patents, other research groups have not been forthcoming in funding research dollars on these patented antitumorous plant chemicals.

    In 2002, however, a well-known research group in North Carolina discovered three new casearins in the leaves and stems of guacatonga that the Japanese had not (and, obviously, hadn't patented). They named the new chemicals casearvestrin A, B and C, and published their first study in February, 2002, stating: "All three compounds displayed promising bioactivity, both in cytotoxicity assays against a panel of tumor cell lines and in antifungal assays . . ." Their research tested the new plant chemicals against human lung, colon and ovarian tumor cells and indicated all three compounds had toxicity to cancer cells in very small amounts. This research was supported by a grant from the National Cancer Institute, National Institutes of Health (NCI) and performed by a non-profit biotech company, a large pharmaceutical company and a major university. The NCI has also performed research in-house on clerodane diterpenoids found in another Casearia plant species documenting the antitumor properties of its novel diterpenoids and another university research group has documented the anticancerous properties of this class of chemicals in a Casearia plant from the Madagascar rainforest as well. It will be interesting to see if this diversified group will actually develop these chemicals into new effective chemotherapeutic agents; their research is ongoing.

    All other research on the chemicals and activities of guacatonga has been performed by Brazilian research groups over the years. The first published toxicity study with rats indicated no toxicity with an ethanol extract of the leaves at 1840 mg per kg. This research group, at the University of Sao Paulo, studied the anti-ulcer properties of the plant (based on its long history of use as an effective herbal remedy for ulcers). They published two studies confirming these benefits. The first study, with rats (in 1990), showed that a crude leaf extract reduced the volume of gastric secretion by 42%, but had little effect on pH. The extract also prevented lab-induced acute gastric mucosal injury which was equivalent to the antiulcer drug cimetidine (Tagamet®). Ten years later they published a second rat study, documenting that a crude leaf extract protected the stomach lining without changing gastric pH and sped healing of acetic acid-induced chronic ulcers and H. pylori ulcers.

    Another Brazilian researcher documented that a bark-and-leaf infusion demonstrated pain relieving and mild anti-inflammatory properties in mice. A university researcher followed up on the anti-inflammatory research, publishing in her dissertation that an extract of the leaves was as effective against inflammation in mice as the NSAID drugs Prioxicam® and Meloxicam®. Leaf extracts have also been shown by two research groups to be active against common food poisoning bacteria strains, Bacillus cerus and B. subtilis, but inactive against such other common bacteria as Staphylococcus, Streptoccoccus, and E. coli.


    It will be interesting to see what happens with guacatonga's ongoing cancer research - especially with sarcomas. These types of tumors typically grow very quickly, are resistant to many of the approved cancer drugs, and represent a bleak prognosis for most cancer patients. In the meantime, guacatonga is considered a safe plant and a great natural herbal remedy for ulcers, inflammation, and pain, and will continue to be used as a snakebite remedy throughout the Amazon jungles by the indigenous peoples. Although not widely available in the U.S. market yet, hopefully as more people learn of its beneficial uses, the market demand for it will increase.

    Main Preparation Method: infusion or capsules
    Main Actions (in order):
    anticancerous, antitumorous, antiulcerous, antivenin, anti-inflammatory

    Main Uses:

    for cancer (sarcoma, carcinoma, and adenocarcinoma)
    for stomach disorders (ulcers, acid reflux, indigestion, dyspepsia, stomachache)
    as an antivenin for snake, spider, and bee bites and stings
    as a topical analgesic (pain-reliever) and anti-inflammatory for skin diseases, rashes, and wounds
    as a blood purifier and general detoxification
    Properties/Actions Documented by Research:
    analgesic (pain-reliever), antacid, anti-inflammatory, antibacterial, anticancerous, antifungal, antitumorous, antiulcerous, antivenin, gastroprotective (protects the gastric tract)
    Other Properties/Actions Documented by Traditional Use:
    anesthetic, antihemorrhagic (reduces bleeding), antimutagenic (cellular protector), antiseptic, antiviral, astringent, blood cleanser, detoxifier, digestive stimulant, wound healer

    Cautions: none

    Traditional Preparation: Twenty grams of dried leaves are infused in a liter of water and quarter-cup amounts are taken 2-3 times daily with meals as a digestive and anti-ulcer aid. Since most of the chemicals are water soluble, powdered leaves in tablets or capsules (1-2 grams twice daily) can be substituted if desired. The above infusion can also be used topically for wounds, burns, skin rashes, and as a mouth wash after dental work or tooth extractions.

    Contraindications: None known.
    Drug Interactions: None reported.

    Bolivia for blood cleansing, cancer, dental antiseptic mouthwash, inflammation, insect bites, pain, skin diseases, snakebite, tumors, ulcers, wounds, and to stop bleeding
    Brazil for blood cleansing, diarrhea, chest and body pains, eczema, fevers, flu, herpes, inflammation, leprosy, male sexual stimulant, rheumatism, skin diseases, snakebite, syphilis, wounds
    Colombia for skin diseases, snakebite, ulcers, wounds
    India for snakebite
    Peru for diarrhea
    Elsewhere for leprosy, snakebite, wounds

    The above text has been printed from The Healing Power of Rainforest Herbs by Leslie Taylor, copyrighted © 2005
    All rights reserved. No part of this document may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage or retrieval system, including websites, without written permission.

    A complete Technical Data Report is available for this plant.

    † The statements contained herein have not been evaluated by the Food and Drug Administration. The information contained in this plant database file is intended for education, entertainment and information purposes only. This information is not intended to be used to diagnose, prescribe or replace proper medical care. The plant described herein is not intended to treat, cure, diagnose, mitigate or prevent any disease. Please refer to our Conditions of Use for using this plant database file and web site.


    Third-Party Published Research on Guacatonga

    All available third-party research on guacatonga can be found at PubMed. A partial listing of the published research on guacatonga is shown below:

    Cytotoxic & Anticancerous Actions:
    Balunas, M. J., et al. "Relationships between inhibitory activity against a cancer cell line panel, profiles of plants collected, and compound classes isolated in an anticancer drug discovery project." Chem. Biodivers. 2006; 3(8): 897-915.
    Shen, Y. C., et al. "Cytotoxic clerodane diterpenoids from Casearia membranacea." J. Nat. Prod. 2005; 68(11): 1665-8.
    Maistro, E. L., et al. “Evaluation of the genotoxic potential of the Casearia sylvestris extract on HTC and V79 cells by the comet assay.” Toxicol. In Vitro. 2004 Jun; 18(3): 337-42.
    Oberlies, N. H., et al. “Novel bioactive clerodane diterpenoids from the leaves and twigs of Casearia sylvestris.” J. Nat. Prod. 2002; 65(2): 95–99.
    Sai Prakash, C. V., et al. “Structure and stereochemistry of new cytotoxic clerodane diterpenoids from the bark of Casearia lucida from the Madagascar rainforest.” J. Nat. Prod. 2002; 65(2): 100-7.
    Beutler, J. A. “Novel cytotoxic diterpenes from Casearia arborea.” J. Nat. Prod. 2000; 63(5): 657-61.
    Almeida, A. “Antitumor and anti-inflammatory effects of extract from Casearia sylvestris: comparative study with Piroxicam and Meloxicam.” Instituto de Ciencias Biomedicas, University of Sao Paulo (Dissertation, 4/02/99).
    Itokawa, H., et al. “Antitumor substances from South American plants.” J. Pharmacobio. Dyn. 1992; 15(1): S-2-.
    Morita, H., et al. “Structures and cytotoxic activity relationship of casearins, new clerodane diterpenes from Casearia sylvestris Sw.” Chem. Pharm. Bull. (Tokyo) 1991 Dec; 39(3): 693–97.
    Itokawa, H., et al. “New antitumor principles, casearins A–F, for Casearia sylvestris Sw. (Flacourtiaceae).” Chem. Pharm. Bull. (Tokyo) 1990; 38(12): 3384–88.
    Itokawa, H., et al. “Isolation of diterpenes as antitumor agents from plants.” Patent—Japan Kokai Tokyo Koho–01 1989; 149, 779: 6pp.
    Itokawa, H., et al. “Antitumor principles from Casearia sylvestris Sw. (Flacourtiaceae), structure elucidation of new clerodane diterpenes by 2-D NMR spectroscopy.” Chem. Pharm. Bull. (Tokyo) 1988 March; 36(4): 1585–88.

    Antiulcer & Antacid Actions:
    Esteves, I., et al. “Gastric antiulcer and anti-inflammatory activities of the essential oil from Casearia sylvestris Sw.” J. Ethnopharmacol. 2005 Oct; 101(1-3): 191-6.
    Sertie, J. A., et al. “Antiulcer activity of the crude extract from the leaves of Casearia slyvestris.” Pharmaceutical Biol. 2000; 38(2): 112–19.
    Basile, A. C., et al. “Pharmacological assay of Casearia sylvestris. I: Preventive anti-ulcer activity and toxicity of the leaf crude extract.” J. Ethnopharmacol. 1990; 30(2): 185–97.

    Neuroprotective Actions:
    da Silva, A. C., et al. "Inhibition of NTPDase, 5'-nucleotidase, Na+/K+-ATPase and acetylcholinesterase activities by subchronic treatment with Casearia sylvestris." Phytomedicine. 2006; 13(7): 509-14.

    Antivenin Actions:
    Raslan, D.S., et al. “Anti-PLA2 action test of Casearia sylvestris Sw.” Boll. Chim. Farm. 2002 Nov-Dec; 141(6): 457-60.
    Borges, M., et al. “Neutralization of proteases from Bothrops snake venoms by the aqueous extract from Casearia sylvestris (Flacourtiaceae).” Toxicon 2001; 39(12): 1863–69.
    Borges, M., et al. “Effects of aqueous extract of Casearia sylvestris (Flacourtiaceae) on actions of snake and bee venoms and on activity of phospholipases A(2).” Comp. Biochem. Physiol. B. 2000 Sep 1; 127(1): 21–30.
    Borges, M., et al. “Partial purification of Casearia sylvestris Sa. extract and its anti-PLA2 Action.” Comp. Biochem. Physiol. Ser. B. 2000; 127b(1): 21–30.
    Ruppelt, B. M., et al. “Pharmacological screening of plants recommended by folk medicine as antisnake venom—I. Analgesic and anti-inflammatory activities.” Mem. Inst. Oswaldo Cruz 1991; 86: 203–05.

    Anti-inflammatory & Pain-Relieving Actions:
    Silva, F.B., et al. “Natural medicaments in endodontics—a comparative study of the anti-inflammatory action.” Pesqui. Odontol. Bras. 2004 Apr-Jun; 18(2): 174-9.
    Almeida, A. “Antitumor and anti-inflammatory effects of extract from Casearia sylvestris: comparative study with Piroxicam and Meloxicam.” Instituto de Ciencias Biomedicas, University of Sao Paulo (Dissertation, 4/02/99).

    Antimicrobial, Antiparasitic, & Insecticidal Actions:
    de Mesquita, M. L.,et al. "In vitro antiplasmodial activity of Brazilian Cerrado plants used as traditional remedies." J. Ethnopharmacol. 2006 Sep 23;
    Rodrigues, A. M., et al. "Larvicidal activity of some Cerrado plant extracts against Aedes aegypti." J. Am. Mosq. Control Assoc. 2006 Jun; 22(2): 314-7.
    Mesquita, M.L., et al. “Antileishmanial and trypanocidal activity of Brazilian Cerrado plants.” Mem. Inst. Oswaldo Cruz. 2005 Nov; 100(7): 783-7.
    Espindola, L. S., et al. “Trypanocidal activity of a new diterpene from Casearia sylvestris var. lingua.” Planta Med. 2004; 70(11): 1093-5.
    de Almeida Alves, T. M. “Biological screening of Brazilian medicinal plants.” Mem. Inst. Oswaldo Cruz. 2000 May/Jun; 95(3): 367–73.
    Chiappeta, A. D., et al. “Higher plants with biological activity—plants of Pernambuco. I.” Rev. Inst. Antibiot. 1983; 21(1/2): 43–50.


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    [This Message was Edited on 02/24/2007]



    [ edit | delete ] dgl 02/24/07 09:15 AM

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    Licorice root

    Latin Name: Glycyrrhiza glabra
    Pharmacopeial Name: Liquiritiae radix
    Other Names: Liquorice, Gancao, Glycyrrhiza, sweet root, Yasti-madhu

    OverviewDescriptionChemistry andPharmacologyUsesContraindicationsSide EffectsUse DuringPregnancy and LactationInteractionswith Other DrugsDosage andAdministrationReferencesAdditionalResources


    Licorice is a perennial herb native to the Mediterranean region, central to southern Russia, and Asia Minor to Iran, now widely cultivated throughout Europe, the Middle East, and Asia. The material of commerce comes from wild plants and "semiwild" plants cultivated in the former U.S.S.R., Turkey, Greece, Iran, China, India, Pakistan, Afghanistan, Syria, Italy, and Spain (Bruneton, 1995; Karnick, 1994; Leung and Foster, 1996; Wichtl and Bisset, 1994). In the Chinese pharmacopeia, Glycyrrhiza glabra, G. uralensis, and G. inflata are officially recognized and are the species usually employed in commerce (Tu, 1992). Licorice is one of the most widely used medicinal herbs and is found in numerous traditional formulas (Leung and Foster, 1996). Until about 1000 c.e, licorice was collected in the wild. Its cultivation was first recorded by Piero de Cresenzi of Bologna in the thirteenth century. Cultivated roots are harvested after three to four years of growth. Its genus name, Glycyrrhiza, given by first century Greek physician Dioscorides, comes from glukos (sweet) and riza (root) (Foster and Yue, 1992; Grieve, 1979).

    Licorice is one of the most extensively researched medicinal and food plants (Chandler, 1997). The roots and stolons contain glycyrrhizin (also knows as glycyrrhizic or glycyrrhizinic acid, about 5–9% by weight), a compound that is about 50 times sweeter than sucrose. Commercial extracts usually contain gycyrrhizin in its ammonium salt form. The sweet taste is reduced or lost in an acidic medium (Leung and Foster, 1996; Foster and Tyler, 1999). Licorice extracts are widely used to flavor food and liqueurs. Millions of pounds of licorice are imported into the United States each year, about 90% for use in flavoring tobacco products (Foster and Tyler, 1999).

    Licorice root has been used therapeutically for several thousand years in both Western and Eastern systems of medicine (Bradley, 1992; Leung and Foster, 1996). A chronological, documented summary of its medical uses since 2100 B.C.E. to the present, with correlations to modern pharmacological research, has been published (Gibson, 1978). Its use is first documented in Assyrian clay tablets (ca. 2500 B.C.E.) and Egyptian papyri. It was used in ancient Arabia to treat coughs and to relieve the unwanted effects of laxatives (Bruneton, 1995). Its use in ancient Scythia spread to Greece. Greek natural scientist Theophrastus (ca. 372–287 B.C.E.) reported its use for dry cough, asthma, and all pectoral diseases (Grieve, 1979). Pliny the Elder (ca. 23–79 C.E.) reported licorice cleared the voice and had expectorant and carminative actions (Der Marderosian, 1999). In China, licorice is first mentioned in the Shen Nong Ben Cao Jing (ca. 25 C.E.), reconstructed "materia medica" from lost text attributed to Shen Nong Shi (ca. 3000 B.C.E.) (Foster and Yue, 1992). According to the Chinese pharmacopeia, licorice, either aqueous dry extract or hydroalcoholic fluidextract, is an abirritant, often used in combination with expectorants and antitussives to diminish irritation of the mucous membrane of the pharynx. It also relieves spasms of the gastrointestinal smooth muscle and shows desoxycorticosterone-like action (Tu, 1992). In India, licorice is used in traditional Ayurvedic, Siddha, and Unani medicines (Nadkarni, 1976). The present-day Ayurvedic Pharmacopoeia reports it is expectorant, demulcent, spasmolytic, antinflammatory, an adrenal agent, and a mild laxative (Karnick, 1994). It is also official in the Indian Pharmacopoeia as a demulcent (IP, 1996).

    In Germany, licorice root is licensed as a standard medicinal tea for bronchitis and for chronic gastritis. It is also used in bronchial teas, stomach teas, and laxative teas available only in the pharmacy. Aqueous and alcoholic extracts of licorice root are used in many bronchial, gastrointestinal, liver and bile, and urological preparations. In the United States, licorice root is often a component of demulcent, expectorant, or laxative preparations (dietary supplement and OTC drug) in aqueous infusion, hydroalcoholic fluidextract and tincture, and solid dosage forms. Licorice root and extracts, fluid and solid, are official in the U.S. National Formulary (NF, 1985).

    Many of the modern therapeutic uses of licorice were known in earlier times. Early claims for a broad spectrum of uses for licorice appear to be borne out by modern research (Gibson, 1978). Human studies have investigated efficacy in subacute hepatic failure (Acharya et al., 1993), chronic hepatitis C (Arase et al., 1997), infectious hepatitis (Chang and But, 1986), hemophilia with HIV-1 infection (Mori et al., 1990), and inhibition of HIV replication in patients with AIDS (Hattori et al., 1989). Studies have investigated its effects, in its traditional context as a component of multi-herb formulas, on testosterone secretion in patients with polycystic ovary syndrome (Takahashi et al., 1988), on treating anxiety (Chen et al., 1985), and on gastric and duodenal ulcer (Chang and But, 1986), among others. However, its use in combination formulas cannot determine definitively that the outcome of studies might not also be the result of the effects of other herbs in the formulas, and not attributable to the presence of the licorice alone.

    Licorice preparations have been studied for possible benefits in treating digestive tract ulcers. In one clinical study, licorice root fluidextract was used to treat 100 patients with early peptic ulcer, of which 86 cases had been unresponsive to conventional treatment, at a dose of 15 ml four times daily for six weeks. Positive effects were reported in 90% of the cases, in 22 of which ulcer craters disappeared by X-ray examination and 28 others showed improvement. In subsequent studies, researchers reported that licorice powder at a dosage of 2.5–5.0 g three times daily was more effective than the fluidextract (Chang and But, 1986).

    Reports in the literature of adverse effects of the consumption of excessive amounts of licorice (more than 20 g per day) have raised concerns about the potential for glycyrrhizin in licorice to produce pseudoaldesteronism (excessive levels of aldesterone, a hormone produced by the adrenalcortex) and resulting risks (headache, lethargy, sodium and water retention, hypertension, potassium loss that upsets the sodium-potassium balance, possibly resulting in cardiac problems, including cardiac arrest). The therapeutic uses and risks of licorice has been reviewed by Chandler (1997) and Stormer et al. (1993).

    A deglycyrrhizinated licorice (DGL) preparation has been developed to provide some of the therapeutic benefits of licorice while reducing risk. A DGL preparation efficacy in treating duodenal and gastric ulcers in clinical trials (D'Imperio et al., 1978; Morgan et al., 1982). DGL may be useful in maintenance therapy for patients with gastric ulcers, although its superiority compared to conventional drugs has been questioned. In a two-year comparison study of a DGL product (Caved–S® tablets; two twice per day) and cimetidine, 400 mg at night, 12% (4 of 34) of the DGL subjects had ulcer recurrence compared with 10% (4 of 41) of the drug group in the first year of treatment; in the second year, 29% (9 of 31) for DGL and 25% (8 of 32) for cimetidine (Morgan et al., 1982). After termination of treatment in two years, ulcers recurred rapidly: 2 of 22 DGL patients and 7 of 23 cimetidine patients. The study concluded that long-term maintenance therapy was safe and reasonably effective.

    DGL has shown success in treating duodenal gastric ulcers in clinical trials (D'Imperio et al., 1978; Morgan et al., 1982) although its superiority, compared to conventional drugs like cimetidine, has not been confirmed.

    Glycyrrhizin itself, in a controlled dosage form with other natural products, has been investigated as a therapy for patients with human immuno-deficiency virus (HIV). One clinical study investigated the effects of glycyrrhizin (SNMC: stronger neo-minophagen C) in 42 hemophilia patients with HIV-1 infection. SNMC is made of 0.2% glycyrrhizin, 0.1% cysteine, and 2.0% glycine dissolved in a saline solution. Patients showed improvement in their clinical symptoms (oral candidiasis, lymph node swelling, rash), immunological functions, and liver functions (Mori et al., 1990). A subsequent study investigated the long-term efficacy of SNMC in 84 patients with chronic hepatitis C. Patients received 100 ml intravenously daily for eight weeks and then two to seven times a week for 2 to 16 years. A reduction in serum alanine aminotransferase (ALT) levels was reported in 34 of the 84 patients (35.7%). This trend toward stabilization of ALT levels was statistically significant. Hepatocellular carcinoma (HCC) in 30 patients with normal ALT levels was slightly lower than the 54 remaining patients with higher ALT scores (p=0.08). An increase in blood pressure was noted in 3 of the 84 patients. The authors concluded that the long-term administration of SNMC for chronic HCC is effective in reducing the risk of liver carcinogenesis (Arase et al., 1997).

    The modern therapeutic applications for licorice root are supportable based on its use in well established systems of traditional medicine, on well documented phytochemical investigations, on pharmacological actions reported from in vitro and in vivo studies in animals, and on human clinical studies.

    Pharmacopeial grade licorice root must contain not less than 4% glycyrrhizic acid, calculated on the dried root, and must pass a thin-layer chromatography (TLC) assay to show the presence of glycyrrhetic acid. Its water-soluble extractive content must be not less than 20% (Bruneton, 1995; IP, 1996; Ph.Eur.3, 1998; Ph.Fr.X., 1990; Wichtl and Bisset, 1994). The Japanese Pharmacopoeia also requires not less than 25% dilute ethanol-soluble extractive (JP XII, 1993).



    Licorice root consists of unpeeled, dried roots and stolons of Glycyrrhiza glabra L. [Fam. Fabaceae], and their preparations in effective dosage. The unpeeled roots contain at least 4% glycyrrhizic acid and 25% water-soluble matter. Licorice root also consists of the peeled, dried roots and stolons of G. glabra and their preparations in effective dosage. The peeled roots contain at least 20% water-soluble matter. The root contains flavanone and isoflavanone derivatives, potassium and calcium salts of glycyrrhizic acid, phytosterols, and coumarins.


    Chemistry and Pharmacology

    Licorice root contains triterpenoid saponins (4–24%), mostly glycyrrhizin, a mixture of the potassium and calcium salts of glycyrrhizic acid; flavonoids (1%), mainly the flavanones liquiritin and liquiritigenin, chalcones isoliquiritin, isoliquiritigenin and isoflavonoids (formononetin); amines (1–2%) asparagine, betaine, and choline; amino acids; 3–15% glucose and sucrose; starch (2–30%); polysaccharides (arabinogalactans); sterols (b-sitosterol); coumarins (glycerin); resin; and volatile oils (0.047%) (Bruneton, 1995; Bradley, 1992; Budavari, 1996; Leung and Foster, 1996; List and Hˆrhammer, 1973–1979; Newall et al., 1996; Wichtl and Bisset, 1994). An extensive review of licorice chemistry has been published recently (Tang and Eisenbrand, 1992).

    The Commission E reported that, according to controlled clinical studies, glycyrrhizic acid and the aglycone of glycyrrhizic acid accelerate the healing of gastric ulcers. Secretolytic and expectorant effects have been confirmed in tests on rabbits. In the isolated rabbit ileum, an antispasmodic action has been observed at concentrations of 1:2500–1:5000.

    The British Herbal Compendium reported its actions as anti-inflammatory, expectorant, demulcent, and adrenocorticotropic (Bradley, 1992).

    The pseudo-aldesterone-like effects are generally attributed to the glycyrrhizic acid. New research suggests that the glycyrrhetenic acid, the hydrolytic metabolite of glycyrrhizic acid, is the primary active component that causes inhibition of peripheral metabolism of corticol, which binds to mineralocorticoid receptors in the same way as aldosterone (Heikens et al., 1995).

    Research suggests two hypotheses for licorice's mechanism of action: binding of glycyrretinic acid to mineralocorticoid receptors and blocking the action of 11-beta-hydroxysteroid dehydrogenase. Recent publications suggest that both may be involved, especially with the confirmation that the blocking of the 11-beta-hydroxysteroid dehydrogenase is temporary and that after this occurs, the pseudoaldesteronism is directly related to increased plasma concentration of licorice metabolites and their binding to mineralocorticoid receptors. Glucocorticoids are usually rapidly metabolized into inactive compounds by 11-beta-hydroxysteroid dehydrogenase, thus controlling glucocorticoid access to mineralocorticoid and glucocorticoid receptors. When licorice prevents the inactivation of hydrocortisone, the result is increased glucocorticoid concentration in mineralocorticoid-responsive tissues, thus resulting in glucocorticoids' occupying mineralocorticoid receptors and producing a mineralocorticoid response, as shown by increased sodium retention and hypertension (Chandler, 1997).



    The Commission E approved the internal use of licorice root for catarrhs of the upper respiratory tract and gastric or duodenal ulcers.

    The British Herbal Compendium indicates its use for bronchitis, peptic ulcer, chronic gastritis, rheumatism and arthritis, and adrenocorticoid insufficiency (Bradley, 1992). The German Standard License approves licorice root infusions for loosening mucus, alleviating discharge in bronchitis, and as an adjuvant in treating spasmodic pains of chronic gastritis (Bradley, 1992; Braun et al., 1997; Wichtl and Bisset, 1994). In France, licorice preparations may be used to treat epigastric bloating, impaired digestion, and flatulence (Bruneton, 1995).

    The World Health Organization recognizes no uses for licorice as being supported by clinical data; WHO recognizes the following uses as being described in pharmacopeias and in traditional systems of medicine: demulcent for sore throats; expectorant in treatment of coughs and bronchial catarrh; prophylaxis and treatment of gastric and duodenal ulcers; used in dyspepsia; anti-inflammatory in treating allergic reactions, rheumatism, and arthritis; to prevent liver toxicity; and to treat tuberculosis and adrenocorticoid insufficiency (WHO, 1999).



    Cholestatic liver disorders, liver cirrhosis, hypertonia, hypokalemia, severe kidney insufficiency.


    Side Effects

    On prolonged use and with higher doses, sodium and water retention and potassium loss may occur, accompanied by hypertension, edema, hypokalemia, and, in rare cases, myoglobinuria.

    [Ed. Note: Within several weeks of discontinuing use, any symptoms of hyperaldosteronism disappear (Mantero, 1981).]


    Use During Pregnancy and Lactation

    Not recommended during pregnancy (McGuffin et al., 1997). No restrictions known during lactation.


    Interactions with Other Drugs

    Potassium loss due to other drugs, e.g., thiazide diuretics, can be increased. With potassium loss, sensitivity to digitalis glycosides increases.


    Dosage and Administration

    Unless otherwise prescribed: About 5–15 g per day of cut or powdered root, or dry extracts equivalent to 200–600 mg of glycyrrhizin.

    Succus liquiritiae: 0.5–1.0 ml for catarrhs of the upper respiratory tract, 1.5–3.0 ml for gastric or duodenal ulcers.

    Infusion or decoction: 2–4 g in 150 ml water, after meals three times daily.

    Fluidextract 1:1 (g/ml): 2–4 ml, after meals three times daily.

    Native dry extract 5–6:1 (w/w): 0.33–0.8 g, after meals three times daily.

    DGL tablets (380 mg DGL 4:1): acute cases (gastric or duodenal ulcers): chew 2–4 tablets before each meal; chronic cases: chew 1–2 tablets before meals (Murray and Pizzorno, 1998).

    Duration of administration: Not longer than four to six weeks without medical advice. There is no objection to using licorice root as a flavoring agent up to a maximum daily dosage equivalent to 100 mg glycyrrhizin.



    Acharya, S.K., S. Dasarathy, A. Tandon, Y.K. Joshi, B.N. Tandon. 1993. A preliminary open trial on interferon stimulator (SNMC) derived from Glycyrrhiza glabra in the treatment of subacute hepatic failure. Indian J Med Res 98:69–74.

    Arase, Y. et al. 1997. The long term efficacy of glycyrrhizin in chronic hepatitis C patients. Cancer 79(8):1494–1500.

    Bradley, P.R. (ed.). 1992. British Herbal Compendium, Vol. 1. Bournemouth: British Herbal Medicine Association.

    Braun, R. et al. 1997. Standardzulassungen f r Fertigarzneimittel—Text and Kommentar. Stuttgart: Deutscher Apotheker Verlag.

    Bruneton, J. 1995. Pharmacognosy, Phytochemistry, Medicinal Plants. Paris: Lavoisier Publishing.

    Budavari, S. (ed.). 1996. The Merck Index: An Encyclopedia of Chemicals, Drugs, and Biologicals, 12th ed. Whitehouse Station, N.J.: Merck & Co, Inc.

    Chandler, R.F. Glycyrrhiza Glabra. De Smet, P.A., K. Keller, H‰nsel R., Chandler, R.F. (eds.). 1997. Adverse Effects of Herbal Drugs, Vol. 3. New York: Springer Verlag.

    Chang, H.M. and P.P.H. But (eds.). 1986. Pharmacology and Applications of Chinese Materia Medica, Vol. 1. Philadelphia: World Scientific. 304–316.

    Chen, H.C., M.T. Hsieh, E. Lai. 1985. Studies on the Suanzaorentang in the treatment of anxiety. Psychopharm (Berl) 85(4):486–487.

    Der Marderosian, A. (ed.). 1999. The Review of Natural Products. St. Louis: Facts and Comparisons.

    D'Imperio, N. G.G. Piccari, F. Sarti, et al. 1978. Double-blind trial in duodenal and gastric ulcers. Acta Gastro-Enterologica Belgica 41:427–434.

    Europ‰isches Arzneibuch, 3rd ed., 1st suppl. (Ph.Eur.3). 1998. Stuttgart: Deutscher Apotheker Verlag. 622–623.

    Foster, S. and V.E. Tyler. 1999. Tyler's Honest Herbal: A Sensible Guide to the Use of Herbs and Related Remedies. New York: Haworth Herbal Press. 241–243.

    Foster, S. and C. Yue. 1992. Herbal Emissaries Bringing Chinese Herbs to the West. Rochester, VT: Healing Arts Press. 112–121.

    Gibson, M.R. 1978. Glycyrrhiza in old and new perspectives. Lloydia 41(4):348–354.

    Grieve, M. 1979. A Modern Herbal. New York: Dover Publications, Inc.

    Hattori, T. et al. 1989. Preliminary evidence for inhibitory effect of glycyrrhizin on HIV replication in patients with AIDS. Antiviral Res 11(5–6):255–262.

    Heikens, J., E. Fliers, E. Endert, M. Ackermans, G. van Montfrans. 1995. Liquorice-induced hypertension—a new understanding of an old disease: case report and brief review. Neth J Med 47(5):230–234.

    Indian Pharmacopoeia, Vol. 1. (IP). 1996. Delhi: Government of India Ministry of Health and Family Welfare—Controller of Publications. 440–442.

    Japanese Pharmacopoeia, 12th ed. (JP XII). 1993. Tokyo: Government of Japan Ministry of Health and Welfare—Yakuji Nippo, Ltd. 130–133.

    Karnick, C.R. 1994. Pharmacopoeial Standards of Herbal Plants, Vols. 1–2. Delhi: Sri Satguru Publications. Vol. 1:158–159; Vol. 2:86.

    Leung, A.Y. and S. Foster. 1996. Encyclopedia of Common Natural Ingredients Used in Food, Drugs, and Cosmetics, 2nd ed. New York: John Wiley & Sons, Inc.

    List, P.H. and L. Hˆrhammer (eds.). 1973–1979. Hagers Handbuch der Pharmazeutischen Praxis, Vols. 1–7. New York: Springer Verlag.

    Mantero, F. 1981. Exogenous mineralocorticoid-like disorders. Clin Endocrinol Metab 10(3): 465–478.

    McGuffin, M., C. Hobbs, R. Upton, A. Goldberg. 1997. American Herbal Product Association's Botanical Safety Handbook. Boca Raton: CRC Press.

    Morgan, A.G., W.A. McAdam, C. Pacsoo, A. Darnborough. 1982. Comparison between cimetidine and Caved-S in the treatment of gastric ulceration, and subsequent maintenance therapy. Gut 23(6):545–551.

    Murray, M. and J. Pizzorno. 1998. Encyclopedia of Natural Medicine. Rocklin, CA: Prima Publishing. 817.

    Mori, K. et al. 1990. Effects of glycyrrhizin (SNMC: stronger neo-minophagen C) in hemophilia patients with HIV-1 infection. Tohoku J Exp Med 162(2):183–193.

    Nadkarni, K.M. 1976. Indian Materia Medica. Bombay: Popular Prakashan. 582–584.

    National Formulary (NF), 16th ed. 1985. Washington, D.C.: American Pharmaceutical Association.

    Newall, C.A., L.A. Anderson, J.D. Phillipson. 1996. Herbal Medicines: A Guide for Health-Care Professionals. London: The Pharmaceutical Press.

    PharmacopÈe FranÁaise Xe …dition (Ph.Fr.X.). 1983–1990. Moulins-les-Metz: Maisonneuve S.A.

    Ph.Eur.3. See Europ‰isches Arzneibuch.

    Stormer, F.C., R. Reistad, J. Alexander. 1993. Glycyrrhizic acid in liquorice—evaluation of health hazard. Food and Chemical Toxicology 31(4):303–312.

    Takahashi, K. et al. 1988. Effect of a traditional herbal medicine (shakuyaku-kanzo-to) on testosterone secretion in patients with polycystic ovary syndrome detected by ultrasound. Nippon Sanka Fujinka Gakkai Zasshi 40(6):789–792.

    Tang, W. and G. Eisenbrand. 1992. Chinese Drugs of Plant Origin: Chemistry, Pharmacology, and Use in Traditional and Modern Medicine. New York: Springer Verlag.

    Tu, G. (ed.). 1992. Pharmacopoeia of the People's Republic of China (English Edition 1992). Beijing: Guangdong Science and Technology Press. 118–119.

    WHO. See World Health Organization.

    Wichtl, M. and N.G. Bisset (eds.). 1994. Herbal Drugs and Phytopharmaceuticals. Stuttgart: Medpharm Scientific Publishers.

    World Health Organization. 1999. "Radix Glycyrrhizae." WHO Monographs on Selected Medicinal Plants, Vol. 1. Geneva: World Health Organization. 183–194.


    Additional Resources

    Bardhan, K.D., D.C. Cumberland, R.A. Dixon, C.D. Holdsworth. 1976. Deglycyrrhizinated liquorice in gastric ulcer: a double blind controlled study. Gut 17(5):397.

    Bensky, D. and A. Gamble.1993. Chinese Herbal Medicine. Seattle: Eastland Press, Inc. 323–325.

    British Pharmaceutical Codex (BPC). 1973. London: The Pharmaceutical Press.

    British Pharmacopoeia (BP). 1988. (With subsequent Addenda up to 1992.) London: Her Majesty's Stationery Office.

    Deutsches Arzneibuch, 9th ed. (DAB 9). 1986. Stuttgart: Deutscher Apotheker Verlag.

    Deutscher Arzneimittel-Codex (DAC). 1986. Stuttgart: Deutscher Apotheker Verlag.

    Engqvist, A., F. von Feilitzen, E. Pyke, H. Reichard. 1973. Double-blind trial of deglycyrrhizinated liquorice in gastric ulcer. Gut 14(9):711–715.

    Hartke, K. and E. Mutschler (eds.). 1988. DAB 9—Kommentar: Deutsches Arzneibuch 9, Vol. 3. Stuttgart: Wissenschaftliche Verlagsgesellschaft. 3187–3192.

    Ming, O. (ed.). 1989. Chinese-English Manual of Common-Used In Traditional Chinese Medicine. Hong Kong: Joint Publishing (H.K.) Co., Ltd. 127–129.

    Morton, J.F. 1977. Major Medicinal Plants: Botany, Culture and Uses. Springfield, IL: Charles C. Thomas. 155–158.

    Pharmacopoeia Helvetica, 7th ed. Vol. 1–4. (Ph.Helv.VII). 1987. Bern: Office Central FÈdÈral des ImprimÈs et du MatÈriel.

    Rees, W.D., J. Rhodes, J.E. Wright, L.F. Stamford, A. Bennett. 1979. Effect of deglycyrrhizinated liquorice on gastric mucosal damage by aspirin. Scand J Gastroenterol 14(5):605–607.

    Reynolds, J.E.F. (ed.). 1989. Martindale: The Extra Pharmacopoeia, 29th ed. London: The Pharmaceutical Press.

    Trease, G.E. and W.C. Evans. 1989. Trease and Evans' Pharmacognosy, 13th ed. London; Philadelphia: BailliËre Tindall.

    Yen, K.Y. 1992. The Illustrated Chinese Materia Medica—Crude and Prepared. Taipei: SMC Publishing, Inc. 42.



    This material was adapted from The Complete German Commission E Monographs—Therapeutic Guide to Herbal Medicines. M. Blumenthal, W.R. Busse, A. Goldberg, J. Gruenwald, T. Hall, C.W. Riggins, R.S. Rister (eds.) S. Klein and R.S. Rister (trans.). 1998. Austin: American Botanical Council; Boston: Integrative Medicine Communications.

    1) The Overview section is new information.

    2) Description, Chemistry and Pharmacology, Uses, Contraindications, Side Effects, Interactions with Other Drugs, and Dosage sections have been drawn from the original work. Additional information has been added in some or all of these sections, as noted with references.

    3) The dosage for equivalent preparations (tea infusion, fluidextract, and tincture) have been provided based on the following example:

    Unless otherwise prescribed: 2 g per day of [powdered, crushed, cut or whole] [plant part]

    Infusion: 2 g in 150 ml of water

    Fluidextract 1:1 (g/ml): 2 ml

    Tincture 1:5 (g/ml): 10 ml

    4) The References and Additional Resources sections are new sections. Additional Resources are not cited in the monograph but are included for research purposes.


    Excerpt from Herbal Medicine: Expanded Commission E Monographs
    Copyright 2000 American Botanical Council
    Published by Integrative Medicine Communications
    Available from the American Botanical Council.

    This material is not intended as a guide to self medication by consumers. The lay reader is advised to discuss the information contained herein with a doctor, pharmacist, nurse or other authorized health care practitioner. Neither the editors nor the publisher accepts any responsibility for the accuracy of the information itself or the consequences from the use or misuse of the information contained herein.

    American Botanical Council, 6200 Manor Rd, Austin, TX 78723
    Phone: 512-926-4900 | Fax: 512-926-2345
    Website: | Email:

    The information on this site is intended for educational purposes only and is not a substitute for the advice of a qualified healthcare professional.



    gastritis 02/24/07 10:49 AM

    i'm not exactly sure what gastritis is.

    is this different than acid reflux?

    i know that nexium is prescribed for that.

    if they are similar then you are in a very larg boat with CFS.

    indigestion and intestinal problems in general i very common in CFS.

    have you been checked for h.pylori. this is the bacteria that causes ulcers.

    there is a big question whether the indigestion part of this is caused by too much acid or too little.

    there is a home test that you can do with baking soda and water to see if you have enough hydrochloric acid in your stomach.

    some people take extra with meals which helps them digest their food better if they are lacking.

    as for things that will simply sooth the stomach, i know of one herb that can found as a tea. it is called Fenugreek. do a google and you will find lots of info. i have ordered some for my sister from:

    also for more immediate relief maybe try a little PeptoBismal.

    if anything here is too jumbled let me know, i'll try to explain a little better.




    CFS/ stomach problems 02/24/07 11:01 AM

    here's an interesting link from this website:

    and here is the baking soda test:

    To perform this test: mix one quarter teaspoon of baking soda in eight ounces of cold water, first thing in the morning, before eating or drinking anything except water. Drink the baking soda solution. Time how long it takes to belch. Time up to five minutes. If you have not belched within five minutes stop timing anyway.

    If your stomach is producing adequate amounts of hydrochloric acid you should probably belch within two to three minutes. Early and repeated belching may be due to excessive stomach acid. Belching results from the acid and baking soda reacting to form carbon dioxide gas. The Heidelberg or Gastrocap tests can be employed for confirmation of the results of this test.



    Protonix 02/24/07 11:18 AM

    Nexium did not do well at all for me and caused bad stomach pains for me. When I was told I had gastritis Protonix 40mg was the only precribed med that helped.

    Occassionally I still have to take a med but now I take OTC Prilosec.

    Good Luck



    Melissa (Lemon Balm) 02/25/07 12:04 AM

    Herbal, very easy to tolerate. I am very sensitive to a lot of medicines, herbals, ect. but this is mild and you could see how you do. It also may help with you sleep. Best of luck.



    gastritis 02/25/07 04:44 AM

    im not sure if what i have is simular to what you have but..

    i have irratable bowel syndrome occasionaly,and take colofac pills only if i get that severe muscle spasm.

    but seeing as im told i have high chrestrol (cant spell it)anyway that fatty stuff in the arteries is 6.5

    i read somewhere last week that eating quaker oats for breakfast lowers the cholesterol,..ive spelt it right now haha i just looked on the packet.

    but i also read that eating cucumber helps,but im sorry i cant think if it was ..helps with memory or cholesterol.but its helping to get that wind out of me.

    im making a right mess of my reply today with this brain fog thingy..sorry.

    but what i want to tell you is.the oats are making my stools softer,and im having cucumber,lettuice with most of my meals (not with the oats though haha).i cut the dark green skin off the cucumber,i dont eat that,as i found my stomach has trouble digesting it.

    anyway,i must be doing something right,as my body is releasing the gas from my tummy,and the cucumber and quaker oats,are to date,the newest food that i have added to my list of foods to try.

    i live in the uk,and ive thrown out all tea bags,and gone onto buying PG tips,tea bags.

    i dont drink much tea when in a flare up,as it makes me feel sickly in my tummy,but love tea when not in a is the only drink my tummy can tolerate when in a flare..oohh and water of course,if its not too cold.

    but what interests me about PG tips is this.

    a advert on tv and written on the tea bags box says that pg tips helps you to focus..

    this is due to the naturally occuring THEANINE, that when combined with the other good properties of tea,helps you to be relaxed yet alert.

    so i thought id just tell you about the three things im currently eating,that are in their own way,helping ease my trapped wind problem,and hopefully helping to make me alert,and relaxed,and hopefully my cholesterol might sort itself out too.

    i dont want my doctor to perscribe cholesterol fighting pills for me (he said he will if my next blood test still shows high cholesterol).

    the reason i dont want pills is,if that pill is added to my current perscription list,its going to look bad on any medical i might have to have in the future,when applying for jobs.i most definately wont get a job if theres a hint im a future heart attack case.

    so im hoping that food stuff will lower that for me naturally.

    kind regards




    gastritis 02/25/07 05:35 AM

    I was d'xd with gastritis, via stomach scoping, several years ago. Took Prevacid double dose for 9 weeks & was on a super strict diet before my stomach got back to normal.

    When mine flares is get real nauseated & super dizzy to the point I feel like I'm gonna pass out sitting down.

    Been taking Prilosec OTC off & on, but reached a point it stopped helping. I'm now on Protonix 40 MG. Stomach was in a bad flare & it only took a week of taking Protonix to shut it down :).

    I have food allergies & try to avoid foods that set it off, but it's not always possible.



    Hi Shirl517... (Some Info on Gastitis) 02/25/07 07:21 AM

    Since gastritis (inflammation/irritation of stomach lining) is a condition that has many causes.

    Gastritis can brief and sudden (Acute Gastritis) such as an upset stomach following the use of an aspirin or alcohol.

    There is also longer-lasting condition (Chronic Gastritis) a type of bacteria that infects the stomach -- Helicobacter pylori -- may lead to chronic gastritis.

    There may also be special conditions or illness which are the reason for the gastritis.

    What did your physician say was the cause of you gastritis?

    Gastritis is associated with various medications, medical and surgical conditions, physical stresses, social habits, chemicals, and infections.

    Some causes of gastritis are:

    Cancer/chemotherapy medications

    Corrosives (acids or lye)
    Alcohol of various types
    Swallowing foreign bodies (paper clips or pins)

    Physical stress (in people who are critically ill or injured)
    After medical procedures (such as endoscopy)
    After an operation to remove part of the stomach
    After radiation treatment for cancer
    Autoimmune diseases
    Pernicious anemia
    Chronic vomiting

    Viral infections
    Fungal (yeast) infections
    Parasites and worms

    Have you spoken with you physician about a different medication? He/she can probably prescribe something that you will better tolerate.

    Feel better,

    Karen :)


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  3. Catseye

    Catseye Member

    Hi, have you been using digestive enzymes with meals? Get a good one with protease and pancreatin in it. They'll help break down the food so it won't irritate you so much. I can't eat meal without them. At least, not yet.

    best wishes,

  4. PVLady

    PVLady New Member

    I also have a very sensitive digestive system. Last November had my gallbladder removed (had gallstones) and hoped it would cure me. I am better but not cured yet.

    You might want to have a ultrasound of your gallbladder to make sure you don't have gallstones. My surgeon said many doctors fail to diagnose them because the symptoms can mimic gastritic.

    Two meds that helped me the most:

    Carafate - Really coats your stomach and digestive system.
    (this is a very old drug that works. My allergist told me about it).

    Nexium - Proton Pump Inhibitor - reduces acids in stomach.

    If you have Gastric Reflux try to sleep with your upper body slightly elevated to prevent reflux during night.

    Have you been tested for H Pylori? That is a breath test, very easy.

    There are some great websites with diets to follow for gastritis.